Shannon D. Shields

Affiliations: 
Neuroscience Genentech, San Francisco, CA, United States 
Area:
Pain
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"Shannon Shields"
Mean distance: 13.92 (cluster 6)
 
SNBCP

Parents

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Allan I. Basbaum grad student 1999-2007 UCSF
 (Anatomical and functional analysis of aquaporin I in the nociceptive and olfactory sensory systems.)
Stephen G. Waxman post-doc 2009-2014 Yale

Collaborators

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Sven-Eric Jordt collaborator 2001-2012
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Publications

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Shields SD, Butt RP, Dib-Hajj SD, et al. (2015) Oral administration of PF-01247324, a subtype-selective Nav1.8 blocker, reverses cerebellar deficits in a mouse model of multiple sclerosis. Plos One. 10: e0119067
Shields SD. (2014) Conditional deletion of HCN2 from primary afferents uncovers the heterogeneity of inflammatory hypersensitivity. Pain. 155: 1051-2
González-Fernández C, Fernández-Martos CM, Shields SD, et al. (2014) Wnts are expressed in the spinal cord of adult mice and are differentially induced after injury. Journal of Neurotrauma. 31: 565-81
Shields SD, Cheng X, Uçeyler N, et al. (2012) Sodium channel Na(v)1.7 is essential for lowering heat pain threshold after burn injury. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 32: 10819-32
Shields SD, Ahn HS, Yang Y, et al. (2012) Nav1.8 expression is not restricted to nociceptors in mouse peripheral nervous system. Pain. 153: 2017-30
Shields SD, Cheng X, Gasser A, et al. (2012) A channelopathy contributes to cerebellar dysfunction in a model of multiple sclerosis. Annals of Neurology. 71: 186-94
Shields SD, Waxman SG. (2011) Cerebellar dysfunction in multiple sclerosis: in the blink of an eye. Multiple Sclerosis (Houndmills, Basingstoke, England). 17: 1152-4
Shields SD. (2011) Intracellular pH in primary somatosensory neurons. Neuroscience Letters. 501: 1-3
Shields SD, Moore KD, Phelps PE, et al. (2010) Olfactory ensheathing glia express aquaporin 1. The Journal of Comparative Neurology. 518: 4329-41
Shields SD, Cavanaugh DJ, Lee H, et al. (2010) Pain behavior in the formalin test persists after ablation of the great majority of C-fiber nociceptors. Pain. 151: 422-9
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