Amber Lockridge
Affiliations: | University of Minnesota, Twin Cities, Minneapolis, MN |
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"Amber Lockridge"Mean distance: 106866 (cluster 6) | S | N | B | C | P |
Parents
Sign in to add mentorLiLian Yuan | research assistant | UMN | |
Emilyn Alejandro | grad student | 2015- | University of Minnesota Medical School |
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Publications
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Mohan R, Jo S, Da Sol Chung E, et al. (2021) Pancreatic β-Cell O-GlcNAc Transferase Overexpression Increases Susceptibility to Metabolic Stressors in Female Mice. Cells. 10 |
Mohan R, Jo S, Lockridge A, et al. (2021) OGT Regulates Mitochondrial Biogenesis and Function via Diabetes Susceptibility Gene Pdx1. Diabetes |
Akhaphong B, Baumann DC, Beetch M, et al. (2021) Placental mTOR-Complex1 regulates fetal programming of obesity and insulin resistance in mice. Jci Insight |
Essawy A, Jo S, Beetch M, et al. (2021) O-linked N-Acetylglucosamine Transferase (OGT) regulates pancreatic α-cell function in mice. The Journal of Biological Chemistry. 100297 |
Lockridge A, Gustafson E, Wong A, et al. (2021) Acute D-Serine Co-Agonism of β-Cell NMDA Receptors Potentiates Glucose-Stimulated Insulin Secretion and Excitatory β-Cell Membrane Activity. Cells. 10 |
Jo S, Lockridge A, Mohan R, et al. (2020) Translational Factor eIF4G1 Regulates Glucose Homeostasis and Pancreatic β-Cell Function. Diabetes |
Jo S, Lockridge A, Mohan R, et al. (2020) Translational Factor eIF4G1 Regulates Glucose Homeostasis and Pancreatic β-Cell Function. Diabetes |
Lockridge A, Jo S, Gustafson E, et al. (2020) Islet O-GlcNAcylation Is Required for Lipid Potentiation of Insulin Secretion through SERCA2. Cell Reports. 31: 107609 |
Jo S, Lockridge A, Alejandro EU. (2019) eIF4G1 and carboxypeptidase E axis dysregulation in O-GlcNAc transferase-deficient pancreatic β cells contributes to hyperproinsulinemia in mice. The Journal of Biological Chemistry |
Alejandro E, Baumann D, Wong A, et al. (2019) Placental mTOR insufficiency during pregnancy predisposes female offspring to obesity-induced insulin resistance Placenta. 83: e12 |