Joseph L. Kissil
Affiliations: | Cell and Molecular Biology | University of Pennsylvania, Philadelphia, PA, United States |
Area:
Cell Biology, OncologyGoogle:
"Joseph Kissil"
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Publications
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Shepard A, Kissil JL. (2020) The use of non-traditional models in the study of cancer resistance-the case of the naked mole rat. Oncogene. 39: 5083-5097 |
Hoxha S, Shepard A, Troutman S, et al. (2020) YAP-mediated recruitment of YY1 and EZH2 represses transcription of key cell cycle regulators. Cancer Research |
Cui Y, Groth S, Troutman S, et al. (2019) The NF2 tumor suppressor merlin interacts with Ras and RasGAP, which may modulate Ras signaling. Oncogene |
Cui Y, Groth S, Troutman S, et al. (2019) The NF2 tumor suppressor merlin interacts with Ras and RasGAP, which may modulate Ras signaling. Oncogene |
Kota S, Hou S, Guerrant W, et al. (2018) A novel three-dimensional high-throughput screening approach identifies inducers of a mutant KRAS selective lethal phenotype. Oncogene |
Chinthalapudi K, Mandati V, Zheng J, et al. (2018) Lipid binding promotes the open conformation and tumor-suppressive activity of neurofibromin 2. Nature Communications. 9: 1338 |
Lyons J, Brubaker DK, Ghazi PC, et al. (2018) Integrated in vivo multiomics analysis identifies p21-activated kinase signaling as a driver of colitis. Science Signaling. 11 |
Carper MB, Troutman S, Byrd KM, et al. (2018) Abstract 1032: Novel mouse models of high-risk HPV-related oral cancers Cancer Research. 78: 1032-1032 |
Moleirinho S, Hoxha S, Mandati V, et al. (2017) Regulation of localization and function of the transcriptional co-activator YAP by angiomotin. Elife. 6 |
Blakeley JO, Bakker A, Barker A, et al. (2017) The path forward: 2015 International Children's Tumor Foundation conference on neurofibromatosis type 1, type 2, and schwannomatosis. American Journal of Medical Genetics. Part A |