Gustavo C. Ferreira

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"Gustavo Ferreira"
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Publications

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Schuck PF, Ferreira GC, McKenna MC. (2020) Recent advances in the pathophysiology of inherited metabolic diseases. International Journal of Developmental Neuroscience : the Official Journal of the International Society For Developmental Neuroscience
Antonini R, Scaini G, Michels M, et al. (2019) Effects of omega-3 fatty acids supplementation on inflammatory parameters after chronic administration of L-tyrosine. Metabolic Brain Disease
Belenguer P, Duarte JMN, Schuck PF, et al. (2019) Mitochondria and the Brain: Bioenergetics and Beyond. Neurotoxicity Research
Carvalho-Silva M, Gomes LM, Gomes ML, et al. (2019) Omega-3 fatty acid supplementation can prevent changes in mitochondrial energy metabolism and oxidative stress caused by chronic administration of L-tyrosine in the brain of rats. Metabolic Brain Disease
McKenna MC, Schuck PF, Ferreira GC. (2018) Fundamentals of CNS energy metabolism and alterations in lysosomal storage diseases. Journal of Neurochemistry
Scaini G, Tonon T, Moura de Souza CF, et al. (2018) Evaluation of plasma biomarkers of inflammation in patients with maple syrup urine disease. Journal of Inherited Metabolic Disease
Gomes LM, Scaini G, Carvalho-Silva M, et al. (2018) Antioxidants Reverse the Changes in the Cholinergic System Caused by L-Tyrosine Administration in Rats. Neurotoxicity Research
Taschetto L, Scaini G, Zapelini HG, et al. (2017) Acute and long-term effects of intracerebroventricular administration of α-ketoisocaproic acid on oxidative stress parameters and cognitive and noncognitive behaviors. Metabolic Brain Disease
Streck EL, De Prá SDT, Ferro PR, et al. (2017) Role of antioxidant treatment on DNA and lipid damage in the brain of rats subjected to a chemically induced chronic model of tyrosinemia type II. Molecular and Cellular Biochemistry
Carvalho-Silva M, Gomes LM, Scaini G, et al. (2017) Omega-3 fatty acid supplementation decreases DNA damage in brain of rats subjected to a chemically induced chronic model of Tyrosinemia type II. Metabolic Brain Disease
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