Efrain Cepeda-Prado

Affiliations: 
University of Magdeburg, Magdeburg, Sachsen-Anhalt, Germany 
Area:
Neurophysiology, synaptic plasticity, neuroprotection
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"Efrain Cepeda-Prado"
Mean distance: 15.2 (cluster 11)
 
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Publications

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Cepeda-Prado EA, Khodaie B, Quiceno GD, et al. (2021) Calcium-Permeable AMPA Receptors Mediate Timing-Dependent LTP Elicited by Low Repeat Coincident Pre- and Postsynaptic Activity at Schaffer Collateral-CA1 Synapses. Cerebral Cortex (New York, N.Y. : 1991)
Nuriel T, Angulo SL, Khan U, et al. (2017) Neuronal hyperactivity due to loss of inhibitory tone in APOE4 mice lacking Alzheimer's disease-like pathology. Nature Communications. 8: 1464
Angulo SL, Orman R, Neymotin SA, et al. (2017) Tau and amyloid-related pathologies in the entorhinal cortex have divergent effects in the hippocampal circuit. Neurobiology of Disease
Edelmann E, Cepeda-Prado E, Leßmann V. (2017) Coexistence of Multiple Types of Synaptic Plasticity in Individual Hippocampal CA1 Pyramidal Neurons. Frontiers in Synaptic Neuroscience. 9: 7
Edelmann E, Cepeda-Prado E, Franck M, et al. (2015) Theta Burst Firing Recruits BDNF Release and Signaling in Postsynaptic CA1 Neurons in Spike-Timing-Dependent LTP. Neuron. 86: 1041-54
Cepeda-Prado E, Popp S, Khan U, et al. (2012) R6/2 Huntington's disease mice develop early and progressive abnormal brain metabolism and seizures. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 32: 6456-67
Lopez-Tobón A, Cepeda-Prado E, Cardona-Gómez GP. (2009) Decrease of Tau hyperphosphorylation by 17β estradiol requires sphingosine kinase in a glutamate toxicity model. Neurochemical Research. 34: 2206-14
Cepeda-Prado E, López-Tobón A, García-Segura LM, et al. (2008) 17 β-estradiol decreases the expression and association of kinases responsible of Tau hyperphosphorylation Colombia Medica. 39: 38-45
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