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Michael R. Due

Affiliations: 
Anesthesia Indiana University School of Medicine, Indianapolis, IN, United States 
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"Michael Due"
Mean distance: 15.17 (cluster 11)
 		SNBCP
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Klaudiusz Weiss grad student 2010 Mount Sinai School of Medicine
 (Extrinsic inputs exert generalized actions on the feeding CPG of Aplysia.)
Fletcher A. White post-doc 2010-2013 Indiana University School of Medicine

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Publications

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Due MR, Wang Y, Barry MA, et al. (2022) Convergent effects of neuropeptides on the feeding central pattern generator of . Journal of Neurophysiology
Xie JY, Chew LA, Yang X, et al. (2016) Sustained relief of ongoing experimental neuropathic pain by a CRMP2 peptide aptamer with low abuse potential. Pain. 157: 2124-2140
Jing J, Alexeeva V, Chen SA, et al. (2015) Functional Characterization of a Vesicular Glutamate Transporter in an Interneuron That Makes Excitatory and Inhibitory Synaptic Connections in a Molluscan Neural Circuit. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 35: 9137-49
Due MR, Yang XF, Allette YM, et al. (2014) Carbamazepine potentiates the effectiveness of morphine in a rodent model of neuropathic pain. Plos One. 9: e107399
Allette YM, Due MR, Wilson SM, et al. (2014) Identification of a functional interaction of HMGB1 with Receptor for Advanced Glycation End-products in a model of neuropathic pain. Brain, Behavior, and Immunity. 42: 169-77
Due MR, Park J, Zheng L, et al. (2014) Acrolein involvement in sensory and behavioral hypersensitivity following spinal cord injury in the rat. Journal of Neurochemistry. 128: 776-86
King AM, Yang XF, Wang Y, et al. (2012) Identification of the benzyloxyphenyl pharmacophore: a structural unit that promotes sodium channel slow inactivation. Acs Chemical Neuroscience. 3: 1037-49
Due MR, Piekarz AD, Wilson N, et al. (2012) Neuroexcitatory effects of morphine-3-glucuronide are dependent on Toll-like receptor 4 signaling. Journal of Neuroinflammation. 9: 200
Piekarz AD, Due MR, Khanna M, et al. (2012) CRMP-2 peptide mediated decrease of high and low voltage-activated calcium channels, attenuation of nociceptor excitability, and anti-nociception in a model of AIDS therapy-induced painful peripheral neuropathy. Molecular Pain. 8: 54
Feldman P, Due MR, Ripsch MS, et al. (2012) The persistent release of HMGB1 contributes to tactile hyperalgesia in a rodent model of neuropathic pain. Journal of Neuroinflammation. 9: 180
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