Ladislav Tauc

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"Ladislav Tauc"
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Fossier P, Blanchard B, Ducrocq C, et al. (1999) Nitric oxide transforms serotonin into an inactive form and this affects neuromodulation. Neuroscience. 93: 597-603
Fossier P, Tauc L, Baux G. (1999) Calcium transients and neurotransmitter release at an identified synapse. Trends in Neurosciences. 22: 161-6
Fossier P, Diebler MF, Mothet JP, et al. (1998) Control of the calcium concentration involved in acetylcholine release and its facilitation: an additional role for synaptic vesicles? Neuroscience. 85: 85-91
Mothet JP, Fossier P, Meunier FM, et al. (1998) Cyclic ADP-ribose and calcium-induced calcium release regulate neurotransmitter release at a cholinergic synapse of Aplysia. The Journal of Physiology. 507: 405-14
Mothet JP, Fossier P, Schirar A, et al. (1996) Opposite effects of nitric oxide on identified inhibitory and excitatory cholinergic synapses of Aplysia californica. Physiological Research / Academia Scientiarum Bohemoslovaca. 45: 177-83
Poulain B, De Paiva A, Deloye F, et al. (1996) Differences in the multiple step process of inhibition of neurotransmitter release induced by tetanus toxin and botulinum neurotoxins type A and B at Aplysia synapses. Neuroscience. 70: 567-76
Mothet JP, Fossier P, Tauc L, et al. (1996) NO decreases evoked quantal ACh release at a synapse of Aplysia by a mechanism independent of Ca2+ influx and protein kinase G. The Journal of Physiology. 493: 769-84
Mothet JP, Fossier P, Tauc L, et al. (1996) Opposite actions of nitric oxide on cholinergic synapses: which pathways? Proceedings of the National Academy of Sciences of the United States of America. 93: 8721-6
Meulemans A, Mothet JP, Schirar A, et al. (1995) A nitric oxide synthase activity is involved in the modulation of acetylcholine release in Aplysia ganglion neurons: a histological, voltammetric and electrophysiological study. Neuroscience. 69: 985-95
Fossier P, Baux G, Tauc L. (1994) N- and P-type Ca2+ channels are involved in acetylcholine release at a neuroneuronal synapse: only the N-type channel is the target of neuromodulators. Proceedings of the National Academy of Sciences of the United States of America. 91: 4771-5
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