Bernhard E. Flucher, Ph.D.

Affiliations: 
Innsbruck Medical University, Innsbruck, Tirol, Austria 
Area:
Calcium channels
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"Bernhard Flucher"
Mean distance: 17.94 (cluster 46)
 
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Publications

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Fernández-Quintero ML, El Ghaleb Y, Tuluc P, et al. (2021) Structural determinants of voltage-gating properties in calcium channels. Elife. 10
Rufenach B, Christy D, Flucher BE, et al. (2020) Multiple Sequence Variants in STAC3 Affect Interactions with Ca1.1 and Excitation-Contraction Coupling. Structure (London, England : 1993)
Coste de Bagneaux P, von Elsner L, Bierhals T, et al. (2020) A homozygous missense variant in CACNB4 encoding the auxiliary calcium channel beta4 subunit causes a severe neurodevelopmental disorder and impairs channel and non-channel functions. Plos Genetics. 16: e1008625
Kaplan MM, Flucher BE. (2019) Postsynaptic Ca1.1-driven calcium signaling coordinates presynaptic differentiation at the developing neuromuscular junction. Scientific Reports. 9: 18450
El Ghaleb Y, Campiglio M, Flucher BE. (2019) Correcting the R165K substitution in the first voltage-sensor of Ca1.1 right-shifts the voltage-dependence of skeletal muscle calcium channel activation. Channels (Austin, Tex.)
Flucher BE, Campiglio M. (2018) STAC proteins: The missing link in skeletal muscle EC coupling and new regulators of calcium channel function. Biochimica Et Biophysica Acta. Molecular Cell Research
Campiglio M, Kaplan MM, Flucher BE. (2018) STAC3 incorporation into skeletal muscle triads occurs independent of the dihydropyridine receptor. Journal of Cellular Physiology
Costé de Bagneaux P, Campiglio M, Benedetti B, et al. (2018) Role of putative voltage-sensor countercharge D4 in regulating gating properties of Ca1.2 and Ca1.3 calcium channels. Channels (Austin, Tex.)
Kaplan MM, Sultana N, Benedetti A, et al. (2018) Calcium Influx and Release Cooperatively Regulate AChR Patterning and Motor Axon Outgrowth during Neuromuscular Junction Formation. Cell Reports. 23: 3891-3904
Campiglio M, Costé de Bagneaux P, Ortner NJ, et al. (2018) STAC proteins associate to the IQ domain of CaV1.2 and inhibit calcium-dependent inactivation. Proceedings of the National Academy of Sciences of the United States of America
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