John F. Wesseling
Affiliations: | Universidad de Navarra, Pamplona, Navarra, Spain |
Area:
Synapses, Short-term PlasticityWebsite:
http://www.cima.es/labs-en/cellular-neurobiology/summary/1Google:
"John Wesseling"Mean distance: 13.25 (cluster 11) | S | N | B | C | P |
Children
Sign in to add trainee| Kashif Mahfooz | grad student | 2010-2014 | CIMA |
| David G. Litvin | post-doc | 2018-2020 | Instituto de Neurociencias de Alicante |
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Publications
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| Conde-Dusman MJ, Dey PN, Elía-Zudaire Ó, et al. (2021) Control of protein synthesis and memory by GluN3A-NMDA receptors through inhibition of GIT1/mTORC1 assembly. Elife. 10 |
| Feldmeyer D, Wesseling JF, Sjöström PJ. (2020) Editorial: Methods for Synaptic Interrogation. Frontiers in Synaptic Neuroscience. 12: 23 |
| Wesseling JF. (2019) Considerations for Measuring Activity-Dependence of Recruitment of Synaptic Vesicles to the Readily Releasable Pool. Frontiers in Synaptic Neuroscience. 11: 32 |
| Wesseling JF, Phan S, Bushong EA, et al. (2019) Sparse force-bearing bridges between neighboring synaptic vesicles. Brain Structure & Function |
| Raja MK, Preobraschenski J, Del Olmo-Cabrera S, et al. (2019) Elevated synaptic vesicle release probability in synaptophysin/gyrin family quadruple knockouts. Elife. 8 |
| Raja MK, Preobraschenski J, Olmo-Cabrera SD, et al. (2019) Author response: Elevated synaptic vesicle release probability in synaptophysin/gyrin family quadruple knockouts Elife |
| Pérez-Otaño I, Larsen RS, Wesseling JF. (2016) Emerging roles of GluN3-containing NMDA receptors in the CNS. Nature Reviews. Neuroscience |
| Mahfooz K, Marco S, Martínez-Turrillas R, et al. (2016) GluN3A promotes NMDA spiking by enhancing synaptic transmission in Huntington's disease models. Neurobiology of Disease |
| Mahfooz K, Singh M, Renden R, et al. (2016) A Well-Defined Readily Releasable Pool with Fixed Capacity for Storing Vesicles at Calyx of Held. Plos Computational Biology. 12: e1004855 |
| Wesseling JF, Pérez-Otaño I. (2015) Modulation of GluN3A expression in Huntington disease: a new n-methyl-D-aspartate receptor-based therapeutic approach? Jama Neurology. 72: 468-73 |