Seiko Makino

Affiliations: 
Wellcome Trust Centre for Human Genetics University of Oxford, Oxford, United Kingdom 
Google:
"Seiko Makino"
Mean distance: 17.46 (cluster 11)
 
SNBCP

Parents

Sign in to add mentor
Ben Fairfax grad student 2009-2012 Oxford
BETA: Related publications

Publications

You can help our author matching system! If you notice any publications incorrectly attributed to this author, please sign in and mark matches as correct or incorrect.

Gilchrist JJ, Makino S, Naranbhai V, et al. (2022) Natural Killer cells demonstrate distinct eQTL and transcriptome-wide disease associations, highlighting their role in autoimmunity. Nature Communications. 13: 4073
Al-Mossawi H, Yager N, Taylor CA, et al. (2019) Context-specific regulation of surface and soluble IL7R expression by an autoimmune risk allele. Nature Communications. 10: 4575
Kasela S, Kisand K, Tserel L, et al. (2017) Pathogenic implications for autoimmune mechanisms derived by comparative eQTL analysis of CD4+ versus CD8+ T cells. Plos Genetics. 13: e1006643
Naranbhai V, Fairfax BP, Makino S, et al. (2015) Genomic modulators of gene expression in human neutrophils. Nature Communications. 6: 7545
Westra HJ, Arends D, Esko T, et al. (2015) Cell Specific eQTL Analysis without Sorting Cells. Plos Genetics. 11: e1005223
Naranbhai V, Fairfax BP, Makino S, et al. (2015) Genomic modulators of gene expression in human neutrophils Nature Communications. 6
Wong D, Lee W, Humburg P, et al. (2014) Genomic mapping of the MHC transactivator CIITA using an integrated ChIP-seq and genetical genomics approach. Genome Biology. 15: 494
Fairfax BP, Humburg P, Makino S, et al. (2014) Innate immune activity conditions the effect of regulatory variants upon monocyte gene expression. Science (New York, N.Y.). 343: 1246949
Plant K, Fairfax BP, Makino S, et al. (2014) Fine mapping genetic determinants of the highly variably expressed MHC gene ZFP57. European Journal of Human Genetics : Ejhg. 22: 568-71
Westra HJ, Peters MJ, Esko T, et al. (2013) Systematic identification of trans eQTLs as putative drivers of known disease associations. Nature Genetics. 45: 1238-43
See more...