Leo Studach, Ph.D.
Affiliations: | 2010 | Basic Medical Sciences | Purdue University, West Lafayette, IN, United States |
Area:
Molecular Biology, Genetics, Virology BiologyGoogle:
"Leo Studach"Mean distance: 19430.2
Parents
Sign in to add mentor| Ourania M. Andrisani | grad student | 2010 | Purdue | |
| (Mechanisms leading to hepatitis B virus X-protein mediated hepatocyte transformation.) | ||||
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Publications
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| Liang H, Studach L, Hullinger RL, et al. (2014) Down-regulation of RE-1 silencing transcription factor (REST) in advanced prostate cancer by hypoxia-induced miR-106b~25. Experimental Cell Research. 320: 188-99 |
| Studach LL, Menne S, Cairo S, et al. (2012) Subset of Suz12/PRC2 target genes is activated during hepatitis B virus replication and liver carcinogenesis associated with HBV X protein. Hepatology (Baltimore, Md.). 56: 1240-51 |
| Wang WH, Studach LL, Andrisani OM. (2011) Proteins ZNF198 and SUZ12 are down-regulated in hepatitis B virus (HBV) X protein-mediated hepatocyte transformation and in HBV replication. Hepatology (Baltimore, Md.). 53: 1137-47 |
| Andrisani OM, Studach L, Merle P. (2011) Gene signatures in hepatocellular carcinoma (HCC). Seminars in Cancer Biology. 21: 4-9 |
| Studach L, Wang WH, Weber G, et al. (2010) Polo-like kinase 1 activated by the hepatitis B virus X protein attenuates both the DNA damage checkpoint and DNA repair resulting in partial polyploidy. The Journal of Biological Chemistry. 285: 30282-93 |
| Studach LL, Rakotomalala L, Wang WH, et al. (2009) Polo-like kinase 1 inhibition suppresses hepatitis B virus X protein-induced transformation in an in vitro model of liver cancer progression. Hepatology (Baltimore, Md.). 50: 414-23 |
| Rakotomalala L, Studach L, Wang WH, et al. (2008) Hepatitis B virus X protein increases the Cdt1-to-geminin ratio inducing DNA re-replication and polyploidy. The Journal of Biological Chemistry. 283: 28729-40 |