Thomas W. Gould, Ph.D.
Affiliations: | Salk Institute | Salk Institute for Biological Studies, La Jolla, CA, United States |
Website:
http://www.linkedin.com/pub/thomas-gould/14/4A6/672Google:
"Thomas Gould"Bio:
Oceania University of Medicine, Samoa
Medical Practice industry
September 2007 – September 2009 (2 years 1 month)
Research Scientist RIKEN
Research industry
September 2005 – September 2007 (2 years 1 month)
Mean distance: 18.35 (cluster 3) | S | N | B | C | P |
Parents
Sign in to add mentor| Ronald Oppenheim | grad student | 1996-2002 | Wake Forest School of Medicine | |
| (Also did a postdoc until 2005) | ||||
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Publications
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| Gould TW, Dominguez B, de Winter F, et al. (2019) Glial cells maintain synapses by inhibiting an activity-dependent retrograde protease signal. Plos Genetics. 15: e1007948 |
| Yang J, Dominguez B, de Winter F, et al. (2011) Nestin negatively regulates postsynaptic differentiation of the neuromuscular synapse. Nature Neuroscience. 14: 324-30 |
| Gould TW, Oppenheim RW. (2011) Motor neuron trophic factors: therapeutic use in ALS? Brain Research Reviews. 67: 1-39 |
| An MC, Lin W, Yang J, et al. (2010) Acetylcholine negatively regulates development of the neuromuscular junction through distinct cellular mechanisms. Proceedings of the National Academy of Sciences of the United States of America. 107: 10702-7 |
| de Castro BM, De Jaeger X, Martins-Silva C, et al. (2009) The vesicular acetylcholine transporter is required for neuromuscular development and function. Molecular and Cellular Biology. 29: 5238-50 |
| Gould TW, Yonemura S, Oppenheim RW, et al. (2008) The neurotrophic effects of glial cell line-derived neurotrophic factor on spinal motoneurons are restricted to fusimotor subtypes. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 28: 2131-46 |
| Gould TW, Oppenheim RW. (2007) Synaptic dysfunction in disease and following injury in the developing and adult nervous system: caveats in the choice of therapeutic intervention. Neuroscience and Biobehavioral Reviews. 31: 1073-87 |
| Buss RR, Gould TW, Ma J, et al. (2006) Neuromuscular development in the absence of programmed cell death: phenotypic alteration of motoneurons and muscle. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 26: 13413-27 |
| Gould TW, Buss RR, Vinsant S, et al. (2006) Complete dissociation of motor neuron death from motor dysfunction by Bax deletion in a mouse model of ALS. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 26: 8774-86 |
| Sato N, Sakuma C, Sato Y, et al. (2006) Distinct susceptibility of developing neurons to death following Bax overexpression in the chicken embryo. Cell Death and Differentiation. 13: 435-45 |