Xiang Lynn Cai, Ph.D.
Affiliations: | Physiology | Southern Illnois University |
Area:
affective diseasesGoogle:
"Xiang Cai"Mean distance: 14.74 (cluster 6) | S | N | B | C | P |
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Publications
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| Cai X, Wu M, Zhang Z, et al. (2023) Electroacupuncture alleviated depression-like behaviors in ventromedial prefrontal cortex of chronic unpredictable mild stress-induced rats: Increasing synaptic transmission and phosphorylating dopamine transporter. Cns Neuroscience & Therapeutics |
| Yan S, Xu C, Yang M, et al. (2023) The expression of agmatinase manipulates the affective state of rats subjected to chronic restraint stress. Neuropharmacology. 109476 |
| Ma X, Yang S, Zhang Z, et al. (2022) Rapid and sustained restoration of astrocytic functions by ketamine in depression model mice. Biochemical and Biophysical Research Communications. 616: 89-94 |
| Yang F, Zhu W, Cai X, et al. (2020) Minocycline alleviates NLRP3 inflammasome-dependent pyroptosis in monosodium glutamate-induced depressive rats. Biochemical and Biophysical Research Communications |
| Zhou MH, Sun FF, Xu C, et al. (2018) Modulation of Kalirin-7 Expression by Hippocampal CA1 5-HT Receptors in Spatial Memory Consolidation. Behavioural Brain Research |
| Dong J, Zhou Q, Wei Z, et al. (2017) Protein kinase A mediates scopolamine-induced mTOR activation and an antidepressant response. Journal of Affective Disorders. 227: 633-642 |
| Zhou Q, Dong J, Xu T, et al. (2017) Synaptic potentiation mediated by L-type voltage-dependent calcium channels mediates the antidepressive effects of lateral habenula stimulation. Neuroscience |
| Wei Z, Zhang K, Zhou Q, et al. (2017) Differential Mechanisms underlying Antidepressant Responses of Ketamine and Imipramine. Cns & Neurological Disorders Drug Targets |
| Zhang K, Xu T, Yuan Z, et al. (2016) Essential roles of AMPA receptor GluA1 phosphorylation and presynaptic HCN channels in fast-acting antidepressant responses of ketamine. Science Signaling. 9: ra123 |
| Qi X, Zhang K, Xu T, et al. (2016) Sex Differences in Long-Term Potentiation at Temporoammonic-CA1 Synapses: Potential Implications for Memory Consolidation. Plos One. 11: e0165891 |