Benjamin Segal, M.D.
Affiliations: | Immunology | University of Michigan, Ann Arbor, Ann Arbor, MI |
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"Benjamin Segal"Mean distance: 31.11 (cluster 11)
Cross-listing: Chemistry Tree
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Sign in to add trainee| Patrick Duncker | grad student | 2018 | University of Michigan |
| Jeffrey R Atkinson | post-doc | 2018- | Ohio State University Wexner Medical Center |
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Publications
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| Segal B, Jerome A, Sas A, et al. (2023) Cytokine polarized, alternatively activated bone marrow neutrophils drive axon regeneration. Research Square |
| Zhang Y, Atkinson J, Burd CE, et al. (2023) Biological aging in multiple sclerosis. Multiple Sclerosis (Houndmills, Basingstoke, England). 13524585231204122 |
| Mannion JM, Segal BM, McLoughlin RM, et al. (2023) Respiratory tract Moraxella catarrhalis and Klebsiella pneumoniae can promote pathogenicity of myelin-reactive Th17 cells. Mucosal Immunology |
| Gardner AM, Atkinson JR, Wilkinson NM, et al. (2023) TAM receptor signaling dictates lesion location and clinical phenotype during experimental autoimmune encephalomyelitis. Journal of Neuroimmunology. 375: 578016 |
| Jerome AD, Atkinson JR, McVey Moffatt AL, et al. (2022) Characterization of Zymosan-Modulated Neutrophils With Neuroregenerative Properties. Frontiers in Immunology. 13: 912193 |
| Atkinson JR, Jerome AD, Sas AR, et al. (2022) Biological aging of CNS-resident cells alters the clinical course and immunopathology of autoimmune demyelinating disease. Jci Insight |
| Mockus TE, Munie A, Atkinson JR, et al. (2021) Encephalitogenic and Regulatory CD8 T Cells in Multiple Sclerosis and Its Animal Models. Journal of Immunology (Baltimore, Md. : 1950). 206: 3-10 |
| Kalinski AL, Yoon C, Huffman LD, et al. (2020) Analysis of the immune response to sciatic nerve injury identifies efferocytosis as a key mechanism of nerve debridement. Elife. 9 |
| Sas AR, Carbajal KS, Jerome AD, et al. (2020) A new neutrophil subset promotes CNS neuron survival and axon regeneration. Nature Immunology |
| Xu J, Neal LM, Ganguly A, et al. (2020) Chemokine receptor CXCR3 is required for lethal brain pathology but not pathogen clearance during cryptococcal meningoencephalitis. Science Advances. 6: eaba2502 |