Danwei Huangfu
Affiliations: | Developmental Biology | Sloan-Kettering Institute, New York, NY, United States |
Area:
Stem cells, development, regenerationWebsite:
http://www.mskcc.org/research/lab/danwei-huangfuGoogle:
"Danwei Huangfu"Parents
Sign in to add mentor| Kathryn V. Anderson | grad student | 2005 | Weill Cornell Medical College | |
| (Cilia and Hedgehog signaling during mouse development.) | ||||
| Douglas A. Melton | post-doc | 2005-2010 | Harvard | |
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Publications
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| Rosen BP, Li QV, Cho H, et al. (2023) Parallel genome-scale CRISPR screens distinguish pluripotency and self-renewal. Biorxiv : the Preprint Server For Biology |
| Chen T, Alcorn H, Devbhandari S, et al. (2022) A hypomorphic mutation in Pold1 disrupts the coordination of embryo size expansion and morphogenesis during gastrulation. Biology Open. 11 |
| Vanoli F, Meskauskaite B, Herviou L, et al. (2021) Generation of human embryonic stem cell models to exploit the EWSR1-CREB fusion promiscuity as a common pathway of transformation in human tumors. Oncogene |
| Huangfu D, García-García MJ, Eggenschwiler J. (2021) Kathryn Anderson (1952-2020). Developmental Cell. 56: 257-259 |
| Beer MA, Shigaki D, Huangfu D. (2020) Enhancer Predictions and Genome-Wide Regulatory Circuits. Annual Review of Genomics and Human Genetics |
| Vardhana SA, Arnold PK, Rosen BP, et al. (2019) Glutamine independence is a selectable feature of pluripotent stem cells. Nature Metabolism. 1: 676-687 |
| Li QV, Rosen BP, Huangfu D. (2019) Decoding pluripotency: Genetic screens to interrogate the acquisition, maintenance, and exit of pluripotency. Wiley Interdisciplinary Reviews. Systems Biology and Medicine. e1464 |
| Lee K, Cho H, Rickert RW, et al. (2019) FOXA2 Is Required for Enhancer Priming during Pancreatic Differentiation. Cell Reports. 28: 382-393.e7 |
| Li QV, Dixon G, Verma N, et al. (2019) Genome-scale screens identify JNK-JUN signaling as a barrier for pluripotency exit and endoderm differentiation. Nature Genetics |
| Amin S, Cook B, Zhou T, et al. (2018) Discovery of a drug candidate for GLIS3-associated diabetes. Nature Communications. 9: 2681 |