Lisa J. Pagliari, Ph.D.
Affiliations: | 2001 | Northwestern University, Evanston, IL |
Area:
Immunology, Molecular Biology, Cell BiologyGoogle:
"Lisa Pagliari"Parents
Sign in to add mentor| Richard M. Pope | grad student | 2001 | Northwestern | |
| (Elucidating the molecular mechanisms regulating macrophage survival.) | ||||
BETA: Related publications
See more...
Publications
|
You can help our author matching system! If you notice any publications incorrectly attributed to this author, please sign in and mark matches as correct or incorrect. |
| Tran TM, Temkin V, Shi B, et al. (2009) TNFalpha-induced macrophage death via caspase-dependent and independent pathways. Apoptosis : An International Journal On Programmed Cell Death. 14: 320-32 |
| Bonzon C, Bouchier-Hayes L, Pagliari LJ, et al. (2006) Caspase-2-induced apoptosis requires bid cleavage: a physiological role for bid in heat shock-induced death. Molecular Biology of the Cell. 17: 2150-7 |
| Pagliari LJ, Kuwana T, Bonzon C, et al. (2005) The multidomain proapoptotic molecules Bax and Bak are directly activated by heat. Proceedings of the National Academy of Sciences of the United States of America. 102: 17975-80 |
| Saleh M, Pagliari LJ, Green DR. (2005) Life support: the alpha4 phosphatase subunit in cell survival and apoptosis. Trends in Cell Biology. 15: 285-7 |
| Liu H, Ma Y, Pagliari LJ, et al. (2004) TNF-alpha-induced apoptosis of macrophages following inhibition of NF-kappa B: a central role for disruption of mitochondria. Journal of Immunology (Baltimore, Md. : 1950). 172: 1907-15 |
| Perlman H, Pagliari LJ, Volin MV. (2002) Regulation of apoptosis and cell cycle activity in rheumatoid arthritis. Current Molecular Medicine. 1: 597-608 |
| Perlman H, Pagliari LJ, Nguyen N, et al. (2001) The Fas-FasL death receptor and PI3K pathways independently regulate monocyte homeostasis. European Journal of Immunology. 31: 2421-30 |
| Liu H, Perlman H, Pagliari LJ, et al. (2001) Constitutively activated Akt-1 is vital for the survival of human monocyte-differentiated macrophages. Role of Mcl-1, independent of nuclear factor (NF)-kappaB, Bad, or caspase activation. The Journal of Experimental Medicine. 194: 113-26 |
| Perlman H, Pagliari LJ, Liu H, et al. (2001) Rheumatoid arthritis synovial macrophages express the Fas-associated death domain-like interleukin-1beta-converting enzyme-inhibitory protein and are refractory to Fas-mediated apoptosis. Arthritis and Rheumatism. 44: 21-30 |
| Pagliari LJ, Perlman H, Liu H, et al. (2000) Macrophages require constitutive NF-kappaB activation to maintain A1 expression and mitochondrial homeostasis. Molecular and Cellular Biology. 20: 8855-65 |