William Mattox
Affiliations: | The University of Texas Graduate School of Biomedical Sciences at Houston |
Area:
Molecular BiologyGoogle:
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Li Y, Rao X, Mattox WW, et al. (2015) RNA-Seq Analysis of Differential Splice Junction Usage and Intron Retentions by DEXSeq. Plos One. 10: e0136653 |
Wang S, Wagner EJ, Mattox W. (2013) Half pint/Puf68 is required for negative regulation of splicing by the SR splicing factor Transformer2. Rna Biology. 10: 1396-406 |
Shen M, Mattox W. (2012) Activation and repression functions of an SR splicing regulator depend on exonic versus intronic-binding position. Nucleic Acids Research. 40: 428-37 |
Su S, O'Day D, Wang S, et al. (2012) Analysis of Alternative Splicing in Drosophila Genetic Mosaics Alternative Pre-Mrna Splicing: Theory and Protocols. 474-479 |
Lazareva AA, Roman G, Mattox W, et al. (2007) A role for the adult fat body in Drosophila male courtship behavior. Plos Genetics. 3: e16 |
Qi J, Su S, Mattox W. (2007) The doublesex splicing enhancer components Tra2 and Rbp1 also repress splicing through an intronic silencer. Molecular and Cellular Biology. 27: 699-708 |
Qi J, Su S, McGuffin ME, et al. (2006) Concentration dependent selection of targets by an SR splicing regulator results in tissue-specific RNA processing. Nucleic Acids Research. 34: 6256-63 |
Xu DQ, Mattox W. (2006) Identification of a splicing enhancer in MLH1 using COMPARE, a new assay for determination of relative RNA splicing efficiencies. Human Molecular Genetics. 15: 329-36 |
Unni E, Su S, Zraly CB, et al. (2003) Analysis of a null mutation in the Drosophila splicing regulator Tra2 suggests its function is restricted to sexual differentiation. Genesis (New York, N.Y. : 2000). 37: 76-83 |
Chandler DS, Qi J, Mattox W. (2003) Direct repression of splicing by transformer-2. Molecular and Cellular Biology. 23: 5174-85 |