☰

Xi Du

Affiliations: 
University of Michigan, Ann Arbor, Ann Arbor, MI 
Area:
Neuroscience, Epilepsy, Neurogenesis
Google:
"Xi Du"
Mean distance: (not calculated yet)
 
Tree Publications Grants Similar researchers PubMed Report error

Parents

Sign in to add mentor
Isabelle Deschenes research assistant 2008-2010 Case Western
Jack Parent grad student 2012-

Children

Sign in to add trainee

Collaborators

Sign in to add collaborator
BETA: Related publications

Publications

You can help our author matching system! If you notice any publications incorrectly attributed to this author, please sign in and mark matches as correct or incorrect.

Szabo GG, Du X, Oijala M, et al. (2017) Extended Interneuronal Network of the Dentate Gyrus. Cell Reports. 20: 1262-1268
Du X, Zhang H, Parent JM. (2017) Rabies Tracing of Birthdated Dentate Granule Cells in Rat Temporal Lobe Epilepsy. Annals of Neurology
Du X, Parent J. (2016) Mini-Brains Make Big Leaps for Studying Human Neural Development and Disease. Epilepsy Currents. 16: 402-404
Du X, Parent JM. (2015) Using Patient-Derived Induced Pluripotent Stem Cells to Model and Treat Epilepsies. Current Neurology and Neuroscience Reports. 15: 588
Hoshi M, Du XX, Shinlapawittayatorn K, et al. (2014) Brugada syndrome disease phenotype explained in apparently benign sodium channel mutations. Circulation. Cardiovascular Genetics. 7: 123-31
Shinlapawittayatorn K, Dudash LA, Du XX, et al. (2011) A novel strategy using cardiac sodium channel polymorphic fragments to rescue trafficking-deficient SCN5A mutations. Circulation. Cardiovascular Genetics. 4: 500-9
Shinlapawittayatorn K, Du XX, Liu H, et al. (2011) A common SCN5A polymorphism modulates the biophysical defects of SCN5A mutations. Heart Rhythm : the Official Journal of the Heart Rhythm Society. 8: 455-62
Shinlapawittayatorn K, Du X, Liu H, et al. (2010) Do Sodium Channel α - α Interactions Contribute to Loss-of-Function Observed in Brugada Syndrome? Biophysical Journal. 98: 311a
Shinlapawittayatorn K, Du X, Liu H, et al. (2009) A Common SCN5A Polymorphism Restores the Biophysical Defects of LQT3 Mutations Biophysical Journal. 96: 249a
See more...