Eunee Lee

Affiliations: 
2013- center for synaptic brain dysfuction Institute for Basic Research (IBS), Korea 
Area:
autism
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Parents

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Daesoo Kim grad student 2006-2013 KAIST
Eunjoon Kim post-doc 2013- KAIST
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Publications

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Lee E, Lee S, Shin JJ, et al. (2021) Excitatory synapses and gap junctions cooperate to improve Pv neuronal burst firing and cortical social cognition in Shank2-mutant mice. Nature Communications. 12: 5116
Lee S, Kang H, Jung H, et al. (2021) Gene Dosage- and Age-Dependent Differential Transcriptomic Changes in the Prefrontal Cortex of -Mutant Mice. Frontiers in Molecular Neuroscience. 14: 683196
Kwon Y, Lee SJ, Lee E, et al. (2019) βPix heterozygous mice have defects in neuronal morphology and social interaction. Biochemical and Biophysical Research Communications
Lee H, Shin W, Kim K, et al. (2019) NGL-3 in the regulation of brain development, Akt/GSK3b signaling, long-term depression, and locomotive and cognitive behaviors. Plos Biology. 17: e2005326
Chung C, Ha S, Kang H, et al. (2018) Early Correction of N-Methyl-D-Aspartate Receptor Function Improves Autistic-like Social Behaviors in Adult Shank2 Mice. Biological Psychiatry
Jung H, Park H, Choi Y, et al. (2018) Sexually dimorphic behavior, neuronal activity, and gene expression in Chd8-mutant mice. Nature Neuroscience
Lee S, Lee E, Kim R, et al. (2018) Shank2 Deletion in Parvalbumin Neurons Leads to Moderate Hyperactivity, Enhanced Self-Grooming and Suppressed Seizure Susceptibility in Mice. Frontiers in Molecular Neuroscience. 11: 209
Kim R, Kim J, Chung C, et al. (2018) Cell type-specificdeletion in mice leads to differential synaptic and behavioral phenotypes. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience
Kim DH, Lee B, Park MH, et al. (2016) Molecular mechanism of betaine on hepatic lipid metabolism: Inhibition of FoxO1 binding to PPARg. Journal of Agricultural and Food Chemistry
Zheng YF, Min JS, Kim D, et al. (2016) In Vitro Inhibition of Human UDP-Glucuronosyl-Transferase (UGT) Isoforms by Astaxanthin, β-Cryptoxanthin, Canthaxanthin, Lutein, and Zeaxanthin: Prediction of in Vivo Dietary Supplement-Drug Interactions. Molecules (Basel, Switzerland). 21
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