Pietro Calissano
Affiliations: | Istituto di Neurobiologia |
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| Amadoro G, Latina V, Corsetti V, et al. (2020) N-terminal tau truncation in the pathogenesis of Alzheimer's disease (AD): Developing a novel diagnostic and therapeutic approach. Biochimica Et Biophysica Acta. 1866: 165584 |
| Perini G, Ciasca G, Minelli E, et al. (2019) Dynamic structural determinants underlie the neurotoxicity of the N-terminal tau 26-44 peptide in Alzheimer's disease and other human tauopathies. International Journal of Biological Macromolecules. 141: 278-289 |
| Naletova I, Satriano C, Pietropaolo A, et al. (2019) The Copper(II)-Assisted Connection between NGF and BDNF by Means of Nerve Growth Factor-Mimicking Short Peptides. Cells. 8 |
| Latina V, Caioli S, Zona C, et al. (2018) NGF-Dependent Changes in Ubiquitin Homeostasis Trigger Early Cholinergic Degeneration in Cellular and Animal AD-Model. Frontiers in Cellular Neuroscience. 12: 487 |
| Maino B, Spampinato AG, Severini C, et al. (2018) The trophic effect of nerve growth factor in primary cultures of rat hippocampal neurons is associated to an anti-inflammatory and immunosuppressive transcriptional program. Journal of Cellular Physiology |
| Florenzano F, Veronica C, Ciasca G, et al. (2017) Extracellular truncated tau causes early presynaptic dysfunction associated with Alzheimer's disease and other tauopathies. Oncotarget. 8: 64745-64778 |
| Florenzano F, Veronica C, Ciasca G, et al. (2017) Extracellular truncated tau causes early presynaptic dysfunction associated with Alzheimer's disease and other tauopathies. Oncotarget |
| Latina V, Caioli S, Zona C, et al. (2017) Impaired NGF/TrkA Signaling Causes Early AD-Linked Presynaptic Dysfunction in Cholinergic Primary Neurons. Frontiers in Cellular Neuroscience. 11: 68 |
| Canu N, Pagano I, La Rosa LR, et al. (2017) Association of TrkA and APP Is Promoted by NGF and Reduced by Cell Death-Promoting Agents. Frontiers in Molecular Neuroscience. 10: 15 |
| Severini C, Petrocchi Passeri P, Ciotti MT, et al. (2017) Nerve growth factor derivative NGF61/100 promotes outgrowth of primary sensory neurons with reduced signs of nociceptive sensitization. Neuropharmacology |