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Tyler Tarr

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2016- University of Maryland School of Medicine, Baltimore, MD, United States 
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Stephen D. Meriney grad student 2011-2015 University of Maryland Medical School
Thomas A. Blanpied post-doc 2016-2021 University of Maryland Medical School

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Publications

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Ginebaugh SP, Badawi Y, Laghaei R, et al. (2023) Simulations of active zone structure and function at mammalian NMJs predict that loss of calcium channels alone is not sufficient to replicate LEMS effects. Journal of Neurophysiology
Sarkar D, Kang J, Wassie AT, et al. (2022) Revealing nanostructures in brain tissue via protein decrowding by iterative expansion microscopy. Nature Biomedical Engineering
Ginebaugh SP, Badawi Y, Tarr TB, et al. (2022) Neuromuscular Active Zone Structure and Function in Healthy and Lambert-Eaton Myasthenic Syndrome States. Biomolecules. 12
Held RG, Liu C, Ma K, et al. (2020) Synapse and Active Zone Assembly in the Absence of Presynaptic Ca Channels and Ca Entry. Neuron
Laghaei R, Ma J, Tarr T, et al. (2017) Transmitter release site organization can predict synaptic function at the neuromuscular junction. Journal of Neurophysiology
Meriney SD, Tarr TB, Ojala KS, et al. (2017) Lambert-Eaton myasthenic syndrome: mouse passive-transfer model illuminates disease pathology and facilitates testing therapeutic leads. Annals of the New York Academy of Sciences
Tarr TB, Wipf P, Meriney SD. (2014) Synaptic Pathophysiology and Treatment of Lambert-Eaton Myasthenic Syndrome. Molecular Neurobiology
Tarr TB, Lacomis D, Reddel SW, et al. (2014) Complete reversal of Lambert-Eaton myasthenic syndrome synaptic impairment by the combined use of a K+ channel blocker and a Ca2+ channel agonist. The Journal of Physiology. 592: 3687-96
Tarr TB, Malick W, Liang M, et al. (2013) Evaluation of a novel calcium channel agonist for therapeutic potential in Lambert-Eaton myasthenic syndrome. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 33: 10559-67
Tarr TB, Dittrich M, Meriney SD. (2013) Are unreliable release mechanisms conserved from NMJ to CNS? Trends in Neurosciences. 36: 14-22
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