Thomas Monahan

Affiliations: 
Cardiology University of Maryland School of Medicine, Baltimore, MD, United States 
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Yu D, Gernapudi R, Drucker C, et al. (2019) The myristoylated alanine-rich C kinase substrate differentially regulates kinase interacting with stathmin in vascular smooth muscle and endothelial cells and potentiates intimal hyperplasia formation. Journal of Vascular Surgery
Yu D, Makkar G, Dong T, et al. (2015) MARCKS Signaling Differentially Regulates Vascular Smooth Muscle and Endothelial Cell Proliferation through a KIS-, p27kip1- Dependent Mechanism. Plos One. 10: e0141397
Yu D, Makkar G, Strickland DK, et al. (2015) Myristoylated Alanine-Rich Protein Kinase Substrate (MARCKS) Regulates Small GTPase Rac1 and Cdc42 Activity and Is a Critical Mediator of Vascular Smooth Muscle Cell Migration in Intimal Hyperplasia Formation. Journal of the American Heart Association. 4
Monahan TS, Andersen ND, Martin MC, et al. (2009) MARCKS silencing differentially affects human vascular smooth muscle and endothelial cell phenotypes to inhibit neointimal hyperplasia in saphenous vein. Faseb Journal : Official Publication of the Federation of American Societies For Experimental Biology. 23: 557-64
Monahan TS, Andersen ND, Panossian H, et al. (2007) A novel function for cadherin 11/osteoblast-cadherin in vascular smooth muscle cells: modulation of cell migration and proliferation. Journal of Vascular Surgery. 45: 581-9
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