José-manuel Cancela

Affiliations: 
UPMC Univ Paris 6, France 
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"José-manuel Cancela"
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de Zélicourt A, Fayssoil A, Mansart A, et al. (2023) Two-pore channels (TPCs) acts as a hub for excitation-contraction coupling, metabolism and cardiac hypertrophy signalling. Cell Calcium. 117: 102839
Martucci LL, Launay JM, Kawakami N, et al. (2023) Endolysosomal TPCs regulate social behavior by controlling oxytocin secretion. Proceedings of the National Academy of Sciences of the United States of America. 120: e2213682120
Martucci LL, Cancela JM. (2022) Neurophysiological functions and pharmacological tools of acidic and non-acidic Ca stores. Cell Calcium. 104: 102582
de Zélicourt A, Fayssoil A, Dakouane-Giudicelli M, et al. (2022) CD38-NADase is a new major contributor to Duchenne muscular dystrophic phenotype. Embo Molecular Medicine. e12860
Martucci LL, Amar M, Chaussenot R, et al. (2019) A multiscale analysis in CD38 mice unveils major prefrontal cortex dysfunctions. Faseb Journal : Official Publication of the Federation of American Societies For Experimental Biology. fj201800489R
Cosker F, Cheviron N, Yamasaki M, et al. (2010) The ecto-enzyme CD38 is a nicotinic acid adenine dinucleotide phosphate (NAADP) synthase that couples receptor activation to Ca2+ mobilization from lysosomes in pancreatic acinar cells. The Journal of Biological Chemistry. 285: 38251-9
Bezin S, Charpentier G, Lee HC, et al. (2008) Regulation of nuclear Ca2+ signaling by translocation of the Ca2+ messenger synthesizing enzyme ADP-ribosyl cyclase during neuronal depolarization. The Journal of Biological Chemistry. 283: 27859-70
Bezin S, Fossier P, Cancela JM. (2008) Nucleoplasmic reticulum is not essential in nuclear calcium signalling mediated by cyclic ADPribose in primary neurons Pflugers Archiv European Journal of Physiology. 456: 581-586
Menteyne A, Burdakov A, Charpentier G, et al. (2006) Generation of specific Ca(2+) signals from Ca(2+) stores and endocytosis by differential coupling to messengers. Current Biology : Cb. 16: 1931-7
Jacquard C, Trioulier Y, Cosker F, et al. (2006) Brain mitochondrial defects amplify intracellular [Ca2+] rise and neurodegeneration but not Ca2+ entry during NMDA receptor activation. Faseb Journal : Official Publication of the Federation of American Societies For Experimental Biology. 20: 1021-3
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