Zahid Padamsey
Affiliations: | University of Oxford, Oxford, United Kingdom |
Area:
Long-term potentiation, presynaptic terminal, synaptic plasticity, hippocampusGoogle:
"Zahid Padamsey"Mean distance: 16.28 (cluster 6) | S | N | B | C | P |
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Publications
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| Tong R, Emptage NJ, Padamsey Z. (2020) A two-compartment model of synaptic computation and plasticity. Molecular Brain. 13: 79 |
| Francioni V, Padamsey Z, Rochefort NL. (2019) High and asymmetric somato-dendritic coupling of V1 layer 5 neurons independent of visual stimulation and locomotion. Elife. 8 |
| Padamsey Z, Tong R, Emptage N. (2019) Optical Quantal Analysis Using Ca Indicators: A Robust Method for Assessing Transmitter Release Probability at Excitatory Synapses by Imaging Single Glutamate Release Events. Frontiers in Synaptic Neuroscience. 11: 5 |
| Emptage N, Padamsey Z. (2019) A role for presynaptic NMDA receptors in hippocampal plasticity Ibro Reports. 6: S34-S35 |
| Vasquez-Lopez SA, Turcotte R, Koren V, et al. (2018) Subcellular spatial resolution achieved for deep-brain imaging in vivo using a minimally invasive multimode fiber. Light, Science & Applications. 7: 110 |
| Foster WJ, Taylor HBC, Padamsey Z, et al. (2018) Hippocampal mGluR1-dependent long-term potentiation requires NAADP-mediated acidic store Ca signaling. Science Signaling. 11 |
| Padamsey Z, Foster WJ, Emptage NJ. (2018) Intracellular Ca Release and Synaptic Plasticity: A Tale of Many Stores. The Neuroscientist : a Review Journal Bringing Neurobiology, Neurology and Psychiatry. 1073858418785334 |
| Vasquez-Lopez SA, Turcotte R, Koren V, et al. (2018) Subcellular spatial resolution achieved for deep-brain imaging in vivo using a minimally invasive multimode fiber Light: Science & Applications. 7: 110 |
| Padamsey Z, McGuinness L, Emptage NJ. (2017) Inhibition of lysosomal Ca2+ signalling disrupts dendritic spine structure and impairs wound healing in neurons. Communicative & Integrative Biology. 10: e1344802 |
| Padamsey Z, Tong R, Emptage N. (2017) Glutamate is required for depression but not potentiation of long-term presynaptic function. Elife. 6 |