Mark Connor

University of Sydney, Camperdown, New South Wales, Australia 
Trigeminal sensory neurons, opioid receptors, ion channels, cannabinoids
"Mark Connor"
Mean distance: 14.67 (cluster 11)
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Gillis A, Gondin AB, Kliewer A, et al. (2020) Low intrinsic efficacy for G protein activation can explain the improved side effect profiles of new opioid agonists. Science Signaling. 13
Sachdev S, Banister SD, Santiago M, et al. (2020) Differential activation of G protein-mediated signaling by synthetic cannabinoid receptor agonists. Pharmacology Research & Perspectives. 8: e00566
Sachdev S, Boyd R, Grimsey NL, et al. (2019) Brodifacoum does not modulate human cannabinoid receptor-mediated hyperpolarization of AtT20 cells or inhibition of adenylyl cyclase in HEK 293 cells. Peerj. 7: e7733
Santiago M, Sachdev S, Arnold JC, et al. (2019) Absence of Entourage: Terpenoids Commonly Found in Do Not Modulate the Functional Activity of Δ-THC at Human CB and CB Receptors. Cannabis and Cannabinoid Research. 4: 165-176
Sachdev S, Vemuri K, Banister SD, et al. (2019) In vitro determination of the CB1 efficacy of illicit synthetic cannabinoids. British Journal of Pharmacology
Udoh M, Santiago M, Devenish S, et al. (2019) Cannabichromene is a cannabinoid CB2 receptor agonist. British Journal of Pharmacology
Sreenivasan VKA, W Razali WA, Zhang K, et al. (2017) Development of Bright and Biocompatible Nanoruby and its Application to Background-free Time-gated Imaging of G-protein Coupled Receptors. Acs Applied Materials & Interfaces
Banister SD, Longworth M, Kevin R, et al. (2016) The pharmacology of valinate and tert-leucinate synthetic cannabinoids 5F-AMBICA, 5F-AMB, 5F-ADB, AMB-FUBINACA, MDMB-FUBINACA, MDMB-CHMICA, and their analogues. Acs Chemical Neuroscience
Jorgensen WT, Gulliver DW, Werry EL, et al. (2015) Flexible analogues of WAY-267,464: Synthesis and pharmacology at the human oxytocin and vasopressin 1a receptors. European Journal of Medicinal Chemistry. 108: 730-740
Redmond WJ, Cawston EE, Grimsey NL, et al. (2015) Identification of N-arachidonoyl dopamine as a highly biased ligand at cannabinoid CB1 receptors. British Journal of Pharmacology
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