Claire E. Hulsebosch
Affiliations: | University of Texas Medical Branch, Galveston, TX |
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Children
Sign in to add trainee| Adrianne B. Ondarza | grad student | 2000 | UTMB |
| Bryan C. Hains | grad student | 2001 | UTMB |
| Charles D. Mills | grad student | 2002 | UTMB |
| Huaiyu Tan | grad student | 2007 | UTMB |
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Publications
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| Gwak YS, Hulsebosch CE, Leem JW. (2017) Neuronal-Glial Interactions Maintain Chronic Neuropathic Pain after Spinal Cord Injury. Neural Plasticity. 2017: 2480689 |
| Carter MW, Johnson KM, Lee JY, et al. (2016) Comparison of Mechanical Allodynia and Recovery of Locomotion and Bladder Function by Different Parameters of Low Thoracic Spinal Contusion Injury in Rats. The Korean Journal of Pain. 29: 86-95 |
| Perez-Polo JR, Rea HC, Johnson KM, et al. (2015) Inflammatory cytokine receptor blockade in a rodent model of mild traumatic brain injury. Journal of Neuroscience Research |
| Perez-Polo JR, Rea HC, Johnson KM, et al. (2015) A rodent model of mild traumatic brain blast injury. Journal of Neuroscience Research. 93: 549-61 |
| Hassler SN, Johnson KM, Hulsebosch CE. (2014) Reactive oxygen species and lipid peroxidation inhibitors reduce mechanical sensitivity in a chronic neuropathic pain model of spinal cord injury in rats. Journal of Neurochemistry. 131: 413-7 |
| Gwak YS, Hassler SE, Hulsebosch CE. (2013) Reactive oxygen species contribute to neuropathic pain and locomotor dysfunction via activation of CamKII in remote segments following spinal cord contusion injury in rats. Pain. 154: 1699-708 |
| Robertson CS, Hulsebosch CE. (2013) Studies of mild traumatic brain injury. Journal of Neurotrauma. 30: 687 |
| Perez-Polo JR, Rea HC, Johnson KM, et al. (2013) Inflammatory consequences in a rodent model of mild traumatic brain injury. Journal of Neurotrauma. 30: 727-40 |
| Dewitt DS, Perez-Polo R, Hulsebosch CE, et al. (2013) Challenges in the development of rodent models of mild traumatic brain injury. Journal of Neurotrauma. 30: 688-701 |
| Crown ED, Gwak YS, Ye Z, et al. (2012) Calcium/calmodulin dependent kinase II contributes to persistent central neuropathic pain following spinal cord injury. Pain. 153: 710-21 |