Julie Secombe
Affiliations: | Genetics and Neuroscience | Albert Einstein College of Medicine, New York, New York, United States |
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Yheskel M, Hatch HAM, Pedrosa E, et al. (2024) KDM5-mediated transcriptional activation of ribosomal protein genes alters translation efficiency to regulate mitochondrial metabolism in neurons. Nucleic Acids Research |
Rogers MF, Marshall OJ, Secombe J. (2023) KDM5-mediated activation of genes required for mitochondrial biology is necessary for viability in Drosophila. Development (Cambridge, England) |
Yheskel M, Sidoli S, Secombe J. (2023) Proximity labeling reveals a new in vivo network of interactors for the histone demethylase KDM5. Epigenetics & Chromatin. 16: 8 |
Hatch HAM, Secombe J. (2021) Molecular and cellular events linking variants in the histone demethylase KDM5C to the intellectual disability disorder Claes-Jensen syndrome. The Febs Journal |
Hatch HA, Belalcazar HM, Marshall OJ, et al. (2021) A KDM5-Prospero transcriptional axis functions during early neurodevelopment to regulate mushroom body formation. Elife. 10 |
Belalcazar HM, Hendricks EL, Zamurrad S, et al. (2021) The histone demethylase KDM5 is required for synaptic structure and function at the Drosophila neuromuscular junction. Cell Reports. 34: 108753 |
Drelon C, Rogers MF, Belalcazar HM, et al. (2019) The histone demethylase KDM5 controls developmental timing in by promoting prothoracic gland endocycles. Development (Cambridge, England). 146 |
Chen K, Luan X, Liu Q, et al. (2019) Drosophila Histone Demethylase KDM5 Regulates Social Behavior through Immune Control and Gut Microbiota Maintenance. Cell Host & Microbe |
Drelon C, Belalcazar HM, Secombe J. (2018) The Histone Demethylase KDM5 Is Essential for Larval Growth in . Genetics |
Zamurrad S, Hatch HAM, Drelon C, et al. (2018) A Drosophila Model of Intellectual Disability Caused by Mutations in the Histone Demethylase KDM5. Cell Reports. 22: 2359-2369 |