Richard Anthony Baylis
Affiliations: | University of Virginia, Charlottesville, VA |
Area:
AtherosclerosisGoogle:
"Richard Baylis"Mean distance: (not calculated yet)
BETA: Related publications
See more...
Publications
You can help our author matching system! If you notice any publications incorrectly attributed to this author, please sign in and mark matches as correct or incorrect. |
Karnewar S, Karnewar V, Deaton R, et al. (2023) IL-1β inhibition partially negates the beneficial effects of diet-induced lipid lowering. Biorxiv : the Preprint Server For Biology |
Gole S, Tkachenko S, Masannat T, et al. (2022) Endothelial-to-Mesenchymal Transition in Atherosclerosis: Friend or Foe? Cells. 11 |
Shin J, Tkachenko S, Chaklader M, et al. (2022) Endothelial OCT4 is atheroprotective by preventing metabolic and phenotypic dysfunction. Cardiovascular Research |
Newman AAC, Serbulea V, Baylis RA, et al. (2021) Multiple cell types contribute to the atherosclerotic lesion fibrous cap by PDGFRβ and bioenergetic mechanisms. Nature Metabolism. 3: 166-181 |
Alencar GF, Owsiany KM, K S, et al. (2020) The Stem Cell Pluripotency Genes Klf4 and Oct4 Regulate Complex SMC Phenotypic Changes Critical in Late-Stage Atherosclerotic Lesion Pathogenesis. Circulation |
Mahan S, Liu M, Baylis RA, et al. (2019) Quantitative Analysis of Cellular Composition in Advanced Atherosclerotic Lesions of Smooth Muscle Cell Lineage-Tracing Mice. Journal of Visualized Experiments : Jove |
Hess DL, Kelly-Goss MR, Cherepanova OA, et al. (2019) Perivascular cell-specific knockout of the stem cell pluripotency gene Oct4 inhibits angiogenesis. Nature Communications. 10: 967 |
Newman AA, Baylis RA, Hess DL, et al. (2018) Irradiation abolishes smooth muscle investment into vascular lesions in specific vascular beds. Jci Insight. 3 |
Gomez D, Baylis RA, Durgin BG, et al. (2018) Interleukin-1β has atheroprotective effects in advanced atherosclerotic lesions of mice. Nature Medicine |
Baylis RA, Gomez D, Mallat Z, et al. (2017) The CANTOS Trial: One Important Step for Clinical Cardiology but a Giant Leap for Vascular Biology. Arteriosclerosis, Thrombosis, and Vascular Biology |