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John E Greer, M.D., Ph.D.

Affiliations: 
Anatomy & Neurobiology Virginia Commonwealth University, Richmond, VA, United States 
Area:
Traumatic brain injury
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John T. Povlishock grad student VCU School of Medicine
Kimberle M. Jacobs post-doc 2020-2021 VCU

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Publications

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Harris AC, Jin XT, Greer JE, et al. (2022) Somatostatin interneurons exhibit enhanced functional output and resilience to axotomy after mild traumatic brain injury. Neurobiology of Disease. 171: 105801
Ogino Y, Bernas T, Greer JE, et al. (2021) Axonal injury following mild traumatic brain injury is exacerbated by repetitive insult and is linked to the delayed attenuation of NeuN expression without concomitant neuronal death in the mouse. Brain Pathology (Zurich, Switzerland). e13034
Hånell A, Greer JE, Jacobs KM. (2015) Increased Network Excitability Due to Altered Synaptic Inputs to Neocortical Layer V Intact and Axotomized Pyramidal Neurons after Mild Traumatic Brain Injury. Journal of Neurotrauma. 32: 1590-8
Hånell A, Greer JE, McGinn MJ, et al. (2015) Traumatic brain injury-induced axonal phenotypes react differently to treatment. Acta Neuropathologica. 129: 317-32
Greer JE, HÃ¥nell A, McGinn MJ, et al. (2013) Mild traumatic brain injury in the mouse induces axotomy primarily within the axon initial segment. Acta Neuropathologica. 126: 59-74
Greer JE, Povlishock JT, Jacobs KM. (2012) Electrophysiological abnormalities in both axotomized and nonaxotomized pyramidal neurons following mild traumatic brain injury. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 32: 6682-7
Greer JE, McGinn MJ, Povlishock JT. (2011) Diffuse traumatic axonal injury in the mouse induces atrophy, c-Jun activation, and axonal outgrowth in the axotomized neuronal population. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 31: 5089-105
Reeves TM, Greer JE, Vanderveer AS, et al. (2010) Proteolysis of submembrane cytoskeletal proteins ankyrin-G and αII-spectrin following diffuse brain injury: a role in white matter vulnerability at Nodes of Ranvier. Brain Pathology (Zurich, Switzerland). 20: 1055-68
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