Juan Tamargo
Affiliations: | Institute Pharmacology and Toxicology CSIC, Madrid |
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Sign in to add trainee| Maria I. Martin | grad student | Institute Pharmacology and Toxicology CSIC, Madrid | |
| Ricardo Gómez | grad student | 2002-2011 |
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| Cámara-Checa A, Rubio-Alarcón M, Dago M, et al. (2024) Reply to Benndorff and DiFrancesco: Reliable human HCN4 single-channel recordings using the cell-attached configuration in expression systems. Proceedings of the National Academy of Sciences of the United States of America. 121: e2402992121 |
| Cámara-Checa A, Perin F, Rubio-Alarcón M, et al. (2023) A gain-of-function HCN4 mutant in the HCN domain is responsible for inappropriate sinus tachycardia in a Spanish family. Proceedings of the National Academy of Sciences of the United States of America. 120: e2305135120 |
| Tinaquero D, Crespo-García T, Utrilla RG, et al. (2020) The p.P888L SAP97 polymorphism increases the transient outward current (I) and abbreviates the action potential duration and the QT interval. Scientific Reports. 10: 10707 |
| Pérez-Hernández M, Matamoros M, Alfayate S, et al. (2018) Brugada syndrome trafficking-defective Nav1.5 channels can trap cardiac Kir2.1/2.2 channels. Jci Insight. 3 |
| Tamargo J, Caballero R, Delpón E. (2018) New Therapeutic Approaches for the Treatment of Hyperkalemia in Patients Treated with Renin-Angiotensin-Aldosterone System Inhibitors. Cardiovascular Drugs and Therapy. 32: 99-119 |
| Utrilla RG, Nieto-Marín P, Alfayate S, et al. (2017) Kir2.1-Nav1.5 Channel Complexes Are Differently Regulated than Kir2.1 and Nav1.5 Channels Alone. Frontiers in Physiology. 8: 903 |
| Caballero R, Utrilla RG, Amorós I, et al. (2017) Tbx20 controls the expression of the KCNH2 gene and of hERG channels. Proceedings of the National Academy of Sciences of the United States of America |
| Tamargo J, Ruilope LM. (2017) Investigational calcium channel blockers for the treatment of hypertension. Expert Opinion On Investigational Drugs. 25: 1295-1309 |
| Rivera-Torres J, Calvo CJ, Llach A, et al. (2016) Cardiac electrical defects in progeroid mice and Hutchinson-Gilford progeria syndrome patients with nuclear lamina alterations. Proceedings of the National Academy of Sciences of the United States of America |
| Matamoros M, Pérez-Hernández M, Guerrero-Serna G, et al. (2016) Nav1.5 N-terminal domain binding to α1-syntrophin increases membrane density of human Kir2.1, Kir2.2 and Nav1.5 channels. Cardiovascular Research. 110: 279-90 |