Andrew J. Boreland
Affiliations: | Cell Biology and Neuroscience | Rutgers University, New Brunswick, New Brunswick, NJ, United States |
Area:
Neural Organoids and iPSC-derived MicrogliaGoogle:
"Andrew Boreland"Mean distance: (not calculated yet)
Parents
Sign in to add mentorPeng Jiang | grad student | 2017- | Rutgers, New Brunswick |
Zhiping Pang | grad student | 2017- | Rutgers - Robert Wood Johnson Medical School |
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Publications
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Li X, Liu J, Boreland AJ, et al. (2024) Polygenic risk for alcohol use disorder affects cellular responses to ethanol exposure in a human microglial cell model. Science Advances. 10: eado5820 |
Boreland AJ, Stillitano AC, Lin HC, et al. (2024) Sustained type I interferon signaling after human immunodeficiency virus type 1 infection of human iPSC derived microglia and cerebral organoids. Iscience. 27: 109628 |
Boreland AJ, Stillitano AC, Lin HC, et al. (2023) Dysregulated neuroimmune interactions and sustained type I interferon signaling after human immunodeficiency virus type 1 infection of human iPSC derived microglia and cerebral organoids. Biorxiv : the Preprint Server For Biology |
Gabriel R, Boreland AJ, Pang ZP. (2023) Whole Cell Patch Clamp Electrophysiology in Human Neuronal Cells. Methods in Molecular Biology (Clifton, N.J.). 2683: 259-273 |
Robles DA, Boreland AJ, Pang ZP, et al. (2021) A Cerebral Organoid Connectivity Apparatus to Model Neuronal Tract Circuitry. Micromachines. 12 |
Xu R, Boreland AJ, Li X, et al. (2021) Developing human pluripotent stem cell-based cerebral organoids with a controllable microglia ratio for modeling brain development and pathology. Stem Cell Reports |
Scarnati MS, Boreland AJ, Joel M, et al. (2020) Differential sensitivity of human neurons carrying μ opioid receptor (MOR) N40D variants in response to ethanol. Alcohol (Fayetteville, N.Y.) |
Xu R, Li X, Boreland AJ, et al. (2020) Human iPSC-derived mature microglia retain their identity and functionally integrate in the chimeric mouse brain. Nature Communications. 11: 1577 |
Shah R, Zhu M, Boreland A, et al. (2018) Abstract 5493: Targeted inhibition of glutaminase in GRM1-expressing melanoma cells inhibits cell proliferation by reducing glutamate bioavailability Cancer Research. 78: 5493-5493 |