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Philippe Lory, Ph.D.

Affiliations: 
Département de Physiologie CNRS, Montpellier, Montpellier, Occitanie, France 
Area:
ion channels
Website:
http://www.igf.cnrs.fr/
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"Philippe Lory"
Mean distance: 17.35 (cluster 28)
 		SNBCP
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Parents

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Arnold Schwartz post-doc University of Cincinnati

Children

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Joel P. Baumgart grad student CNRS, Montpellier

Collaborators

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Edward Perez-Reyes collaborator CNRS, Montpellier
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Publications

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Milman A, Ventéo S, Bossu JL, et al. (2021) A sodium background conductance controls the spiking pattern of mouse adrenal chromaffin cells in situ. The Journal of Physiology
Lory P, Nicole S, Monteil A. (2020) Neuronal Cav3 channelopathies: recent progress and perspectives. Pflugers Archiv : European Journal of Physiology. 472: 831-844
Chemin J, Stamenic TT, Cazade M, et al. (2019) A novel phospho-modulatory mechanism contributes to the calcium-dependent regulation of T-type Ca channels. Scientific Reports. 9: 15642
Bouasse M, Impheng H, Servant Z, et al. (2019) Functional expression of CLIFAHDD and IHPRF pathogenic variants of the NALCN channel in neuronal cells reveals both gain- and loss-of-function properties. Scientific Reports. 9: 11791
Chemin J, Siquier-Pernet K, Nicouleau M, et al. (2018) De novo mutation screening in childhood-onset cerebellar atrophy identifies gain-of-function mutations in the CACNA1G calcium channel gene. Brain : a Journal of Neurology
El Alaoui C, Chemin J, Fechtali T, et al. (2017) Modulation of T-type Ca2+ channels by Lavender and Rosemary extracts. Plos One. 12: e0186864
Dorgans K, Salvi J, Bertaso F, et al. (2017) Characterization of the dominant inheritance mechanism of Episodic Ataxia type 2. Neurobiology of Disease
Cazade M, Bidaud I, Lory P, et al. (2017) Activity-dependent regulation of T-type calcium channels by submembrane calcium ions. Elife. 6
Daniil G, Fernandes-Rosa FL, Chemin J, et al. (2016) CACNA1H Mutations Are Associated With Different Forms of Primary Aldosteronism. Ebiomedicine
Daniil G, Fernandes-Rosa FL, Chemin J, et al. (2016) [OP.LB01.12] CACNA1H MUTATIONS ARE ASSOCIATED WITH YOUNG ONSET AND FAMILIAL FORMS OF PRIMARY ALDOSTERONISM. Journal of Hypertension. 34: e39
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