Philippe Lory, Ph.D.
Affiliations: | Département de Physiologie | CNRS, Montpellier, Montpellier, Occitanie, France |
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http://www.igf.cnrs.fr/Google:
"Philippe Lory"Mean distance: 17.35 (cluster 28) | S | N | B | C | P |
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Publications
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Qebibo L, Davakan A, Nesson-Dauphin M, et al. (2024) The characterization of new de novo CACNA1G variants affecting the intracellular gate of Cav3.1 channel broadens the spectrum of neurodevelopmental phenotypes in SCA42ND. Genetics in Medicine : Official Journal of the American College of Medical Genetics. 101337 |
Huang J, Fan X, Jin X, et al. (2024) Structural basis for human Ca3.2 inhibition by selective antagonists. Cell Research |
Lory P. (2024) The good company of T-type calcium channels : Commentary on: "T-type calcium channelosome," a review by N. Weiss and G. W. Zamponi. Pflugers Archiv : European Journal of Physiology. 476: 149-150 |
Monteil A, Guérineau NC, Gil-Nagel A, et al. (2023) New insights into the physiology and pathophysiology of the atypical sodium leak channel NALCN. Physiological Reviews |
Nicole S, Lory P. (2021) New Challenges Resulting From the Loss of Function of Na1.4 in Neuromuscular Diseases. Frontiers in Pharmacology. 12: 751095 |
Guérineau NC, Monteil A, Lory P. (2021) Sodium background currents in endocrine/neuroendocrine cells: Towards unraveling channel identity and contribution in hormone secretion. Frontiers in Neuroendocrinology. 63: 100947 |
Lory P, Nicole S, Monteil A. (2020) Neuronal Cav3 channelopathies: recent progress and perspectives. Pflugers Archiv : European Journal of Physiology. 472: 831-844 |
Chemin J, Stamenic TT, Cazade M, et al. (2019) A novel phospho-modulatory mechanism contributes to the calcium-dependent regulation of T-type Ca channels. Scientific Reports. 9: 15642 |
Bouasse M, Impheng H, Servant Z, et al. (2019) Functional expression of CLIFAHDD and IHPRF pathogenic variants of the NALCN channel in neuronal cells reveals both gain- and loss-of-function properties. Scientific Reports. 9: 11791 |
Chemin J, Siquier-Pernet K, Nicouleau M, et al. (2018) De novo mutation screening in childhood-onset cerebellar atrophy identifies gain-of-function mutations in the CACNA1G calcium channel gene. Brain : a Journal of Neurology |