James W. Crane
Affiliations: | University of Queensland, Saint Lucia, Queensland, Australia |
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Publications
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Crane JW, Holmes NM, Fam J, et al. (2019) Oxytocin increases inhibitory synaptic transmission and blocks the development of long-term potentiation in the lateral amygdala. Journal of Neurophysiology |
Delaney AJ, Crane JW, Holmes NM, et al. (2018) Baclofen acts in the central amygdala to reduce synaptic transmission and impair context fear conditioning. Scientific Reports. 8: 9908 |
Fam J, Holmes N, Delaney A, et al. (2018) Oxytocin receptor activation in the basolateral complex of the amygdala enhances discrimination between discrete cues and promotes configural processing of cues. Psychoneuroendocrinology. 96: 84-92 |
Holmes NM, Crane JW, Tang M, et al. (2017) α2-adrenoceptor-mediated inhibition in the central amygdala blocks fear-conditioning. Scientific Reports. 7: 11712 |
Windels F, Yan S, Stratton PG, et al. (2016) Auditory Tones and Foot-Shock Recapitulate Spontaneous Sub-Threshold Activity in Basolateral Amygdala Principal Neurons and Interneurons. Plos One. 11: e0155192 |
Delaney AJ, Crane JW. (2016) Presynaptic GABAB receptors reduce transmission at parabrachial synapses in the lateral central amygdala by inhibiting N-type calcium channels. Scientific Reports. 6: 19255 |
Windels F, Crane JW, Sah P. (2010) Inhibition dominates the early phase of up-states in the basolateral amygdala. Journal of Neurophysiology. 104: 3433-8 |
Crane JW, Baiquni GP, Sullivan RK, et al. (2009) The C5a anaphylatoxin receptor CD88 is expressed in presynaptic terminals of hippocampal mossy fibres. Journal of Neuroinflammation. 6: 34 |
Crane JW, Windels F, Sah P. (2009) Oscillations in the basolateral amygdala: aversive stimulation is state dependent and resets the oscillatory phase. Journal of Neurophysiology. 102: 1379-87 |
Faber ES, Delaney AJ, Power JM, et al. (2008) Modulation of SK channel trafficking by beta adrenoceptors enhances excitatory synaptic transmission and plasticity in the amygdala. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 28: 10803-13 |