Taitea Dykstra
Affiliations: | Oregon Health and Science University, Portland, OR |
Area:
neuroimmunology, movement disordersGoogle:
"Taitea Dykstra"Mean distance: (not calculated yet)
Parents
Sign in to add mentorShane Antony Liddelow | grad student | 2019-2020 | NYU School of Medicine | |
(Master's Student) | ||||
Jonathan Kipnis | research scientist | 2020-2023 | Washington University in St. Louis, School of Medicine | |
(Bioinformatician) |
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Publications
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Salvador AFM, Dykstra T, Rustenhoven J, et al. (2023) Age-dependent immune and lymphatic responses after spinal cord injury. Neuron |
Brase L, You SF, D'Oliveira Albanus R, et al. (2023) Single-nucleus RNA-sequencing of autosomal dominant Alzheimer disease and risk variant carriers. Nature Communications. 14: 2314 |
Rustenhoven J, Pavlou G, Storck SE, et al. (2023) Age-related alterations in meningeal immunity drive impaired CNS lymphatic drainage. The Journal of Experimental Medicine. 220 |
Goldman DH, Dykstra T, Smirnov I, et al. (2022) Age-associated suppression of exploratory activity during sickness is linked to meningeal lymphatic dysfunction and microglia activation. Nature Aging. 2: 704-713 |
Drieu A, Du S, Storck SE, et al. (2022) Parenchymal border macrophages regulate the flow dynamics of the cerebrospinal fluid. Nature |
Sadick JS, O'Dea MR, Hasel P, et al. (2022) Astrocytes and oligodendrocytes undergo subtype-specific transcriptional changes in Alzheimer's disease. Neuron |
Mazzitelli JA, Smyth LCD, Cross KA, et al. (2022) Cerebrospinal fluid regulates skull bone marrow niches via direct access through dural channels. Nature Neuroscience |
Herz J, Fu Z, Kim K, et al. (2021) GABAergic neuronal IL-4R mediates T cell effect on memory. Neuron. 109: 3609-3618.e9 |
Cugurra A, Mamuladze T, Rustenhoven J, et al. (2021) Skull and vertebral bone marrow are myeloid cell reservoirs for the meninges and CNS parenchyma. Science (New York, N.Y.) |
Da Mesquita S, Herz J, Wall M, et al. (2021) Aging-associated deficit in CCR7 is linked to worsened glymphatic function, cognition, neuroinflammation, and β-amyloid pathology. Science Advances. 7 |