Daniel G Taub
Affiliations: | Harvard Medical School - Boston Children's Hospital |
Area:
neuroscience, somatosensation, cortex, painGoogle:
"Daniel Taub"Mean distance: (not calculated yet)
Parents
Sign in to add mentor| Christopher V. Gabel | grad student | 2013-2018 | Boston University Chobanian & Avedisian School of Medicine (Chemistry Tree) |
| Clifford Woolf | post-doc | 2018- | Harvard Medical School - Boston Children's Hospital |
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Publications
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| Taub DG, Jiang Q, Pietrafesa F, et al. (2023) The Secondary Somatosensory Cortex Gates Mechanical and Thermal Sensitivity. Biorxiv : the Preprint Server For Biology |
| Wimalasena NK, Taub DG, Shim J, et al. (2023) Na1.7 gain-of-function mutation I228M triggers age-dependent nociceptive insensitivity and C-LTMR dysregulation. Experimental Neurology. 114393 |
| Jain A, Gyori BM, Hakim S, et al. (2023) Nociceptor neuroimmune interactomes reveal cell type- and injury-specific inflammatory pain pathways. Biorxiv : the Preprint Server For Biology |
| Tian F, Cheng Y, Zhou S, et al. (2023) Core transcription programs controlling injury-induced neurodegeneration of retinal ganglion cells. Neuron. 111: 444 |
| Tian F, Cheng Y, Zhou S, et al. (2022) Core transcription programs controlling injury-induced neurodegeneration of retinal ganglion cells. Neuron |
| Zhang Z, Roberson DP, Kotoda M, et al. (2022) Automated preclinical detection of mechanical pain hypersensitivity and analgesia. Pain |
| Michoud F, Seehus C, Schönle P, et al. (2020) Epineural optogenetic activation of nociceptors initiates and amplifies inflammation. Nature Biotechnology |
| Taub DG, Awal MR, Gabel CV. (2018) O-GlcNAc Signaling Orchestrates the Regenerative Response to Neuronal Injury in Caenorhabditis elegans. Cell Reports. 24: 1931-1938.e3 |
| Melentijevic I, Toth ML, Arnold ML, et al. (2017) C. elegans neurons jettison protein aggregates and mitochondria under neurotoxic stress. Nature |
| Nawabi H, Belin S, Cartoni R, et al. (2015) Doublecortin-Like Kinases Promote Neuronal Survival and Induce Growth Cone Reformation via Distinct Mechanisms. Neuron |