☰

Samhita S. Rhodes, Ph.D. - Publications

Affiliations: 
Marquette University, Milwaukee, WI, United States 
Area:
Signal Processing, Cardiac Electrophysiology, fMRI
Tree Info Grants Similar researchers PubMed Report error

22 high-probability publications. We are testing a new system for linking publications to authors. You can help! If you notice any inaccuracies, please sign in and mark papers as correct or incorrect matches. If you identify any major omissions or other inaccuracies in the publication list, please let us know.

Year Citation  Score
2015 Rhodes SS, Camara AK, Aldakkak M, Heisner JS, Stowe DF. Stretch-induced increase in cardiac contractility is independent of myocyte Ca2+ while block of stretch channels by streptomycin improves contractility after ischemic stunning. Physiological Reports. 3. PMID 26290532 DOI: 10.14814/Phy2.12486  0.622
2012 Rhodes SS, Camara AK, Heisner JS, Riess ML, Aldakkak M, Stowe DF. Reduced mitochondrial Ca2+ loading and improved functional recovery after ischemia-reperfusion injury in old vs. young guinea pig hearts. American Journal of Physiology. Heart and Circulatory Physiology. 302: H855-63. PMID 22140052 DOI: 10.1152/Ajpheart.00533.2011  0.521
2009 Riess ML, Rhodes SS, Stowe DF, Aldakkak M, Camara AK. Comparison of cumulative planimetry versus manual dissection to assess experimental infarct size in isolated hearts. Journal of Pharmacological and Toxicological Methods. 60: 275-80. PMID 19732842 DOI: 10.1016/J.Vascn.2009.05.012  0.349
2007 Heinen A, Aldakkak M, Stowe DF, Rhodes SS, Riess ML, Varadarajan SG, Camara AK. Reverse electron flow-induced ROS production is attenuated by activation of mitochondrial Ca2+-sensitive K+ channels. American Journal of Physiology. Heart and Circulatory Physiology. 293: H1400-7. PMID 17513497 DOI: 10.1152/Ajpheart.00198.2007  0.406
2007 Camara AK, Aldakkak M, Heisner JS, Rhodes SS, Riess ML, An J, Heinen A, Stowe DF. ROS scavenging before 27 degrees C ischemia protects hearts and reduces mitochondrial ROS, Ca2+ overload, and changes in redox state. American Journal of Physiology. Cell Physiology. 292: C2021-31. PMID 17287367 DOI: 10.1152/Ajpcell.00231.2006  0.539
2007 Heinen A, Camara AK, Aldakkak M, Rhodes SS, Riess ML, Stowe DF. Mitochondrial Ca2+-induced K+ influx increases respiration and enhances ROS production while maintaining membrane potential. American Journal of Physiology. Cell Physiology. 292: C148-56. PMID 16870831 DOI: 10.1152/Ajpcell.00215.2006  0.473
2006 An J, Rhodes SS, Jiang MT, Bosnjak ZJ, Tian M, Stowe DF. Anesthetic preconditioning enhances Ca2+ handling and mechanical and metabolic function elicited by Na+-Ca2+ exchange inhibition in isolated hearts. Anesthesiology. 105: 541-9. PMID 16931987 DOI: 10.1097/00000542-200609000-00018  0.604
2006 Rhodes SS, Camara AK, Ropella KM, Audi SH, Riess ML, Pagel PS, Stowe DF. Ischemia reperfusion dysfunction changes model-estimated kinetics of myofilament interaction due to inotropic drugs in isolated hearts. Biomedical Engineering Online. 5: 16. PMID 16512898 DOI: 10.1186/1475-925X-5-16  0.693
2006 Rhodes SS, Ropella KM, Camara AK, Chen Q, Riess ML, Pagel PS, Stowe DF. Ischemia-reperfusion injury changes the dynamics of Ca2+-contraction coupling due to inotropic drugs in isolated hearts. Journal of Applied Physiology (Bethesda, Md. : 1985). 100: 940-50. PMID 16282437 DOI: 10.1152/Japplphysiol.00285.2005  0.688
2005 Riess ML, Camara AK, Rhodes SS, McCormick J, Jiang MT, Stowe DF. Increasing heart size and age attenuate anesthetic preconditioning in guinea pig isolated hearts. Anesthesia and Analgesia. 101: 1572-6. PMID 16301221 DOI: 10.1213/01.Ane.0000181834.39483.0B  0.382
2005 An J, Camara AK, Riess ML, Rhodes SS, Varadarajan SG, Stowe DF. Improved mitochondrial bioenergetics by anesthetic preconditioning during and after 2 hours of 27 degrees C ischemia in isolated hearts. Journal of Cardiovascular Pharmacology. 46: 280-7. PMID 16116332 DOI: 10.1097/01.Fjc.0000175238.18702.40  0.427
2005 An J, Camara AK, Rhodes SS, Riess ML, Stowe DF. Warm ischemic preconditioning improves mitochondrial redox balance during and after mild hypothermic ischemia in guinea pig isolated hearts. American Journal of Physiology. Heart and Circulatory Physiology. 288: H2620-7. PMID 15653757 DOI: 10.1152/Ajpheart.01124.2004  0.409
2005 Riess ML, Kevin LG, McCormick J, Jiang MT, Rhodes SS, Stowe DF. Anesthetic preconditioning: the role of free radicals in sevoflurane-induced attenuation of mitochondrial electron transport in Guinea pig isolated hearts. Anesthesia and Analgesia. 100: 46-53. PMID 15616050 DOI: 10.1213/01.Ane.0000139346.76784.72  0.351
2004 Camara AK, Chen Q, Rhodes SS, Riess ML, Stowe DF. Negative inotropic drugs alter indexes of cytosolic [Ca(2+)]-left ventricular pressure relationships after ischemia. American Journal of Physiology. Heart and Circulatory Physiology. 287: H667-80. PMID 15059780 DOI: 10.1152/Ajpheart.01142.2003  0.632
2004 Camara AK, Chen Q, An J, Novalija E, Riess ML, Rhodes SS, Stowe DF. Comparison of hyperkalemic cardioplegia with altered [CaCl2] and [MgCl2] on [Ca2+]i transients and function after warm global ischemia in isolated hearts. The Journal of Cardiovascular Surgery. 45: 1-13. PMID 15041929  0.527
2003 Chen Q, Camara AK, Rhodes SS, Riess ML, Novalija E, Stowe DF. Cardiotonic drugs differentially alter cytosolic [Ca2+] to left ventricular relationships before and after ischemia in isolated guinea pig hearts. Cardiovascular Research. 59: 912-25. PMID 14553831 DOI: 10.1016/S0008-6363(03)00524-8  0.619
2003 Rhodes SS, Ropella KM, Camara AK, Chen Q, Riess ML, Stowe DF. How inotropic drugs alter dynamic and static indices of cyclic myoplasmic [Ca2+] to contractility relationships in intact hearts. Journal of Cardiovascular Pharmacology. 42: 539-53. PMID 14508241 DOI: 10.1097/00005344-200310000-00013  0.676
2003 Kevin LG, Novalija E, Riess ML, Camara AK, Rhodes SS, Stowe DF. Sevoflurane exposure generates superoxide but leads to decreased superoxide during ischemia and reperfusion in isolated hearts. Anesthesia and Analgesia. 96: 949-55, table of con. PMID 12651639 DOI: 10.1213/01.Ane.0000052515.25465.35  0.339
2003 Rhodes SS, Ropella KM, Audi SH, Camara AK, Kevin LG, Pagel PS, Stowe DF. Cross-bridge kinetics modeled from myoplasmic [Ca2+] and LV pressure at 17 degrees C and after 37 degrees C and 17 degrees C ischemia. American Journal of Physiology. Heart and Circulatory Physiology. 284: H1217-29. PMID 12531735 DOI: 10.1152/Ajpheart.00816.2002  0.636
2002 Riess ML, Camara AK, Novalija E, Chen Q, Rhodes SS, Stowe DF. Anesthetic preconditioning attenuates mitochondrial Ca2+ overload during ischemia in Guinea pig intact hearts: reversal by 5-hydroxydecanoic acid. Anesthesia and Analgesia. 95: 1540-6, table of con. PMID 12456413 DOI: 10.1097/00000539-200212000-00013  0.592
2002 Riess ML, Camara AK, Chen Q, Novalija E, Rhodes SS, Stowe DF. Altered NADH and improved function by anesthetic and ischemic preconditioning in guinea pig intact hearts. American Journal of Physiology. Heart and Circulatory Physiology. 283: H53-60. PMID 12063274 DOI: 10.1152/Ajpheart.01057.2001  0.431
2002 Riess ML, Novalija E, Heisner JS, Rhodes SS, Stowe DF. Anesthetic Preconditioning Reduces the Rise and Decline in NADH during Ischemia in Guinea Pig Isolated Hearts: Reversal after Mitochondrial KATP Channel Blockade Anesthesiology. 96: A698. DOI: 10.1097/00000542-200209002-00698  0.369
Show low-probability matches.