Year |
Citation |
Score |
2018 |
Kahanovitch U, Cuddapah VA, Pacheco NL, Holt LM, Mulkey DK, Percy AK, Olsen ML. MeCP2 Deficiency Leads to Loss of Glial Kir4.1. Eneuro. 5. PMID 29464197 DOI: 10.1523/ENEURO.0194-17.2018 |
0.61 |
|
2016 |
Nwaobi SE, Cuddapah VA, Patterson KC, Randolph AC, Olsen ML. The role of glial-specific Kir4.1 in normal and pathological states of the CNS. Acta Neuropathologica. PMID 26961251 DOI: 10.1007/S00401-016-1553-1 |
0.659 |
|
2015 |
Cuddapah VA, Sinifunanya EN, Percy AK, Olsen ML. MeCP2 in the regulation of neural activity: Rett syndrome pathophysiological perspectives. Degenerative Neurological and Neuromuscular Disease. 5: 103-116. PMID 32669918 DOI: 10.2147/DNND.S61269 |
0.578 |
|
2015 |
Campbell SL, Robel S, Cuddapah VA, Robert S, Buckingham SC, Kahle KT, Sontheimer H. GABAergic disinhibition and impaired KCC2 cotransporter activity underlie tumor-associated epilepsy. Glia. 63: 23-36. PMID 25066727 DOI: 10.1002/Glia.22730 |
0.688 |
|
2014 |
Cuddapah VA, Robel S, Watkins S, Sontheimer H. A neurocentric perspective on glioma invasion. Nature Reviews. Neuroscience. 15: 455-65. PMID 24946761 DOI: 10.1038/Nrn3765 |
0.721 |
|
2014 |
Cuddapah VA, Pillai RB, Shekar KV, Lane JB, Motil KJ, Skinner SA, Tarquinio DC, Glaze DG, McGwin G, Kaufmann WE, Percy AK, Neul JL, Olsen ML. Methyl-CpG-binding protein 2 (MECP2) mutation type is associated with disease severity in Rett syndrome. Journal of Medical Genetics. 51: 152-8. PMID 24399845 DOI: 10.1136/Jmedgenet-2013-102113 |
0.569 |
|
2013 |
Cuddapah VA, Turner KL, Seifert S, Sontheimer H. Bradykinin-induced chemotaxis of human gliomas requires the activation of KCa3.1 and ClC-3. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 33: 1427-40. PMID 23345219 DOI: 10.1523/Jneurosci.3980-12.2013 |
0.721 |
|
2013 |
Cuddapah VA, Turner KL, Sontheimer H. Calcium entry via TRPC1 channels activates chloride currents in human glioma cells. Cell Calcium. 53: 187-94. PMID 23261316 DOI: 10.1016/J.Ceca.2012.11.013 |
0.744 |
|
2012 |
McFerrin MB, Turner KL, Cuddapah VA, Sontheimer H. Differential role of IK and BK potassium channels as mediators of intrinsic and extrinsic apoptotic cell death. American Journal of Physiology. Cell Physiology. 303: C1070-8. PMID 22992678 DOI: 10.1152/Ajpcell.00040.2012 |
0.688 |
|
2012 |
Cuddapah VA, Habela CW, Watkins S, Moore LS, Barclay TT, Sontheimer H. Kinase activation of ClC-3 accelerates cytoplasmic condensation during mitotic cell rounding. American Journal of Physiology. Cell Physiology. 302: C527-38. PMID 22049206 DOI: 10.1152/Ajpcell.00248.2011 |
0.719 |
|
2011 |
Haas BR, Cuddapah VA, Watkins S, Rohn KJ, Dy TE, Sontheimer H. With-No-Lysine Kinase 3 (WNK3) stimulates glioma invasion by regulating cell volume. American Journal of Physiology. Cell Physiology. 301: C1150-60. PMID 21813709 DOI: 10.1152/Ajpcell.00203.2011 |
0.742 |
|
2011 |
Cuddapah VA, Sontheimer H. Ion channels and transporters [corrected] in cancer. 2. Ion channels and the control of cancer cell migration. American Journal of Physiology. Cell Physiology. 301: C541-9. PMID 21543740 DOI: 10.1152/Ajpcell.00102.2011 |
0.652 |
|
2010 |
Cuddapah VA, Sontheimer H. Molecular interaction and functional regulation of ClC-3 by Ca2+/calmodulin-dependent protein kinase II (CaMKII) in human malignant glioma. The Journal of Biological Chemistry. 285: 11188-96. PMID 20139089 DOI: 10.1074/Jbc.M109.097675 |
0.682 |
|
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