| Year |
Citation |
Score |
| 2021 |
Rochon PL, Theriault C, Rangel Olguin AG, Krishnaswamy A. The cell adhesion molecule Sdk1 shapes assembly of a retinal circuit that detects localized edges. Elife. 10. PMID 34545809 DOI: 10.7554/eLife.70870 |
0.463 |
|
| 2020 |
Rangel Olguin AG, Rochon PL, Krishnaswamy A. New Optical Tools to Study Neural Circuit Assembly in the Retina. Frontiers in Neural Circuits. 14: 44. PMID 32848633 DOI: 10.3389/fncir.2020.00044 |
0.393 |
|
| 2020 |
Trenholm S, Krishnaswamy A. An Annotated Journey through Modern Visual Neuroscience. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 40: 44-53. PMID 31896562 DOI: 10.1523/Jneurosci.1061-19.2019 |
0.659 |
|
| 2018 |
Duan X, Krishnaswamy A, Laboulaye MA, Liu J, Peng YR, Yamagata M, Toma K, Sanes JR. Cadherin Combinations Recruit Dendrites of Distinct Retinal Neurons to a Shared Interneuronal Scaffold. Neuron. PMID 30197236 DOI: 10.1016/J.Neuron.2018.08.019 |
0.731 |
|
| 2018 |
Liu J, Reggiani JDS, Laboulaye MA, Pandey S, Chen B, Rubenstein JLR, Krishnaswamy A, Sanes JR. Tbr1 instructs laminar patterning of retinal ganglion cell dendrites. Nature Neuroscience. PMID 29632360 DOI: 10.1038/S41593-018-0127-Z |
0.734 |
|
| 2017 |
Peng YR, Tran NM, Krishnaswamy A, Kostadinov D, Martersteck EM, Sanes JR. Satb1 Regulates Contactin 5 to Pattern Dendrites of a Mammalian Retinal Ganglion Cell. Neuron. PMID 28781169 DOI: 10.1016/J.Neuron.2017.07.019 |
0.717 |
|
| 2015 |
Krishnaswamy A, Yamagata M, Duan X, Hong YK, Sanes JR. Sidekick 2 directs formation of a retinal circuit that detects differential motion. Nature. 524: 466-70. PMID 26287463 DOI: 10.1038/Nature14682 |
0.754 |
|
| 2015 |
Krishnaswamy A, Yamagata M, Duan X, Hong YK, Sanes JR. Sidekick 2 directs formation of a retinal circuit that detects differential motion Nature. 524: 466-470. DOI: 10.1038/nature14682 |
0.69 |
|
| 2014 |
Duan X, Krishnaswamy A, De la Huerta I, Sanes JR. Type II cadherins guide assembly of a direction-selective retinal circuit. Cell. 158: 793-807. PMID 25126785 DOI: 10.1016/J.Cell.2014.06.047 |
0.721 |
|
| 2014 |
Lilley BN, Krishnaswamy A, Wang Z, Kishi M, Frank E, Sanes JR. SAD kinases control the maturation of nerve terminals in the mammalian peripheral and central nervous systems. Proceedings of the National Academy of Sciences of the United States of America. 111: 1138-43. PMID 24395778 DOI: 10.1073/Pnas.1321990111 |
0.711 |
|
| 2012 |
Krishnaswamy A, Cooper E. Reactive oxygen species inactivate neuronal nicotinic acetylcholine receptors through a highly conserved cysteine near the intracellular mouth of the channel: implications for diseases that involve oxidative stress. The Journal of Physiology. 590: 39-47. PMID 21969449 DOI: 10.1113/jphysiol.2011.214007 |
0.447 |
|
| 2010 |
Campanucci V, Krishnaswamy A, Cooper E. Diabetes depresses synaptic transmission in sympathetic ganglia by inactivating nAChRs through a conserved intracellular cysteine residue. Neuron. 66: 827-34. PMID 20620869 DOI: 10.1016/J.Neuron.2010.06.010 |
0.438 |
|
| 2009 |
Caffery PM, Krishnaswamy A, Sanders T, Liu J, Hartlaub H, Klysik J, Cooper E, Hawrot E. Engineering neuronal nicotinic acetylcholine receptors with functional sensitivity to alpha-bungarotoxin: a novel alpha3-knock-in mouse. The European Journal of Neuroscience. 30: 2064-76. PMID 20128845 DOI: 10.1111/J.1460-9568.2009.07016.X |
0.417 |
|
| 2009 |
Krishnaswamy A, Cooper E. An activity-dependent retrograde signal induces the expression of the high-affinity choline transporter in cholinergic neurons. Neuron. 61: 272-86. PMID 19186169 DOI: 10.1016/j.neuron.2008.11.025 |
0.513 |
|
| 2008 |
Campanucci VA, Krishnaswamy A, Cooper E. Mitochondrial reactive oxygen species inactivate neuronal nicotinic acetylcholine receptors and induce long-term depression of fast nicotinic synaptic transmission. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 28: 1733-44. PMID 18272694 DOI: 10.1523/JNEUROSCI.5130-07.2008 |
0.471 |
|
| 2005 |
Rassadi S, Krishnaswamy A, Pié B, McConnell R, Jacob MH, Cooper E. A null mutation for the alpha3 nicotinic acetylcholine (ACh) receptor gene abolishes fast synaptic activity in sympathetic ganglia and reveals that ACh output from developing preganglionic terminals is regulated in an activity-dependent retrograde manner. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 25: 8555-66. PMID 16162937 DOI: 10.1523/JNEUROSCI.1983-05.2005 |
0.5 |
|
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