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Eduardo E Arteaga-Bracho - Publications

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3/4 high-probability publications. We are testing a new system for linking publications to authors. You can help! If you notice any inaccuracies, please sign in and mark papers as correct or incorrect matches. If you identify any major omissions or other inaccuracies in the publication list, please let us know.

Year Citation  Score
2019 Mehler MF, Petronglo JR, Arteaga-Bracho EE, Gulinello M, Winchester ML, Pichamoorthy N, Young SK, DeJesus CD, Ishtiaq H, Gokhan S, Molero AE. Loss-of-huntingtin in medial and lateral ganglionic lineages differentially disrupts regional interneuron and projection neuron subtypes and promotes Huntington's disease-associated behavioral, cellular and pathological hallmarks. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. PMID 30626701 DOI: 10.1523/Jneurosci.2443-18.2018  0.61
2016 Arteaga-Bracho EE, Gulinello M, Winchester ML, Pichamoorthy N, Petronglo JR, Zambrano AD, Inocencio J, De Jesus CD, Louie JO, Gokhan S, Mehler MF, Molero AE. Postnatal and adult consequences of loss of huntingtin during development: Implications for Huntington's disease. Neurobiology of Disease. PMID 27623015 DOI: 10.1016/J.Nbd.2016.09.006  0.618
2016 Molero AE, Arteaga-Bracho EE, Chen CH, Gulinello M, Winchester ML, Pichamoorthy N, Gokhan S, Khodakhah K, Mehler MF. Selective expression of mutant huntingtin during development recapitulates characteristic features of Huntington's disease. Proceedings of the National Academy of Sciences of the United States of America. PMID 27140644 DOI: 10.1073/Pnas.1603871113  0.607
Low-probability matches (unlikely to be authored by this person)
2014 Chen CH, Fremont R, Arteaga-Bracho EE, Khodakhah K. Short latency cerebellar modulation of the basal ganglia. Nature Neuroscience. 17: 1767-75. PMID 25402853 DOI: 10.1038/nn.3868  0.268
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