Viacheslav O. Nikolaev

2003-2010 Experimental Cardiovascular Research University of Hamburg, Hamburg, Hamburg, Germany 
"Viacheslav Nikolaev"
Mean distance: 14.87 (cluster 6)


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Martin J. Lohse grad student 2003-2010 University of Hamburg
 (PhD in 2005, the Post-Doc)
Julia Gorelik post-doc 2007-2010 University of Hamburg (Physiology Academic Tree)


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Sophie Schobesberger post-doc 2014- University of Hamburg (Physiology Academic Tree)
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Kraft AE, Bork NI, Subramanian H, et al. (2024) Phosphodiesterases 4B and 4D Differentially Regulate cAMP Signaling in Calcium Handling Microdomains of Mouse Hearts. Cells. 13
Lai P, Hille SS, Subramanian H, et al. (2023) Remodelling of cAMP dynamics within the SERCA2a microdomain in heart failure with preserved ejection fraction caused by obesity and type 2 diabetes. Cardiovascular Research
Bork NI, Subramanian H, Kurelic R, et al. (2023) Role of Phosphodiesterase 1 in the Regulation of Real-Time cGMP Levels and Contractility in Adult Mouse Cardiomyocytes. Cells. 12
Kostenis E, Gomeza J, Miess-Tanneberg E, et al. (2023) Reply to: How carvedilol does not activate β-adrenoceptors. Nature Communications. 14: 7867
Lezoualc'h F, Nikolaev VO. (2023) Receptor-Specific Inside-Out cAMP Signaling Regulates Cardiomyocyte Fate. Circulation Research. 133: 924-926
Kwan Z, Nadappuram BP, Leung MM, et al. (2023) Microtubule-Mediated Regulation of βAR Translation and Function in Failing Hearts. Circulation Research
De Jong KA, Ehret S, Heeren J, et al. (2023) Live-cell imaging identifies cAMP microdomains regulating β-adrenoceptor-subtype-specific lipolytic responses in human white adipocytes. Cell Reports. 42: 112433
Benkel T, Zimmermann M, Zeiner J, et al. (2022) How Carvedilol activates β-adrenoceptors. Nature Communications. 13: 7109
Faleeva M, Diakonov I, Srivastava P, et al. (2022) Compartmentation of cGMP Signaling in Induced Pluripotent Stem Cell Derived Cardiomyocytes during Prolonged Culture. Cells. 11
Beneke K, Pavlidou NG, Schäfer A, et al. (2021) GPCR signaling is highly compartmentalized in human cardiomyocytes and severely remodeled in atrial fibrillation. The Journal of General Physiology. 154
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