Elias Aizenman, Ph.D.
Affiliations: | 1989- | Neurobiology | University of Pittsburgh, Pittsburgh, PA, United States |
Area:
Zinc, Potassium Channels, Apoptosis, Redox, NMDA, TOPAWebsite:
http://aizenmanlab.neurobio.pitt.edu/Google:
"Elias Aizenman"Mean distance: 13.23 (cluster 11) | S | N | B | C | P |
Parents
Sign in to add mentorChirstopher H. Price | research assistant | 1980-1981 | Boston University |
George G. Bierkamper | grad student | 1981-1985 | Johns Hopkins |
Elise F. Stanley | grad student | 1981-1985 | Johns Hopkins |
Stuart A. Lipton | post-doc | 1985-1987 | Harvard Medical School |
Children
Sign in to add traineeCollaborators
Sign in to add collaboratorPaul A. Rosenberg | collaborator | 1989- | University of Pittsburgh |
Edwin S. Levitan | collaborator | 2000- | University of Pittsburgh |
Thanos Tzounopoulos | collaborator | 2009- | University of Pittsburgh |
Michael J. Kuhar | collaborator | 1981-1983 | Johns Hopkins |
Ralph Loring | collaborator | 1985-1989 | Harvard Medical School |
Ian J. Reynolds | collaborator | 1989-2000 | University of Pittsburgh |
BETA: Related publications
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Publications
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Krall RF, Moutal A, Phillips MB, et al. (2020) Synaptic zinc inhibition of NMDA receptors depends on the association of GluN2A with the zinc transporter ZnT1. Science Advances. 6 |
Schulien AJ, Yeh CY, Orange BN, et al. (2020) Targeted disruption of Kv2.1-VAPA association provides neuroprotection against ischemic stroke in mice by declustering Kv2.1 channels. Science Advances. 6 |
Yeh CY, Schulien AJ, Molyneaux BJ, et al. (2020) Lessons from Recent Advances in Ischemic Stroke Management and Targeting Kv2.1 for Neuroprotection. International Journal of Molecular Sciences. 21 |
Aizenman E, Loring RH, Reynolds IJ, et al. (2020) The Redox Biology of Excitotoxic Processes: The NMDA Receptor, TOPA Quinone, and the Oxidative Liberation of Intracellular Zinc. Frontiers in Neuroscience. 14: 778 |
XiangWei W, Kannan V, Xu Y, et al. (2019) Heterogeneous clinical and functional features of GRIN2D-related developmental and epileptic encephalopathy. Brain : a Journal of Neurology |
Yeh CY, Ye Z, Moutal A, et al. (2019) Defining the Kv2.1-syntaxin molecular interaction identifies a first-in-class small molecule neuroprotectant. Proceedings of the National Academy of Sciences of the United States of America |
Justice JA, Manjooran DT, Yeh CY, et al. (2018) Molecular neuroprotection induced by zinc-dependent expression of hepatitis C-derived protein NS5A targeting Kv2.1 potassium channels. The Journal of Pharmacology and Experimental Therapeutics |
Yeh CY, Bulas AM, Moutal A, et al. (2017) Targeting a Potassium Channel/Syntaxin Interaction Ameliorates Cell Death in Ischemic Stroke. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience |
Justice JA, Schulien AJ, He K, et al. (2017) Disruption of KV2.1 somato-dendritic clusters prevents the apoptogenic increase of potassium currents. Neuroscience |
Ogden KK, Chen W, Swanger SA, et al. (2017) Molecular Mechanism of Disease-Associated Mutations in the Pre-M1 Helix of NMDA Receptors and Potential Rescue Pharmacology. Plos Genetics. 13: e1006536 |