Stuart A. Lipton

Affiliations: 
Scripps Research Institute, La Jolla, La Jolla, CA, United States 
 Neurosciences University of California, San Diego, La Jolla, CA 
Area:
Neurodegenerative Disease
Website:
https://www.scripps.edu/research/faculty/lipton
Google:
"Stuart Lipton"
Bio:

https://www.scripps.edu/lipton/members.html

Mean distance: 12.18 (cluster 11)
 
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Publications

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Oh CK, Piña-Crespo J, Talantova M, et al. (2023) Reply to: Targeted protein S-nitrosylation of ACE2 inhibits SARS-CoV-2 infection. Nature Chemical Biology. 19: 1306-1308
Oh CK, Nakamura T, Beutler N, et al. (2022) Targeted protein S-nitrosylation of ACE2 inhibits SARS-CoV-2 infection. Nature Chemical Biology
Oh CK, Nakamura T, Beutler N, et al. (2022) Targeted protein S-nitrosylation of ACE2 as potential treatment to prevent spread of SARS-CoV-2 infection. Biorxiv : the Preprint Server For Biology
Trudler D, Sanz-Blasco S, Eisele YS, et al. (2021) α-Synuclein Oligomers Induce Glutamate Release from Astrocytes and Excessive Extrasynaptic NMDAR Activity in Neurons, Thus Contributing to Synapse Loss. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience
Ghatak S, Dolatabadi N, Gao R, et al. (2020) NitroSynapsin ameliorates hypersynchronous neural network activity in Alzheimer hiPSC models. Molecular Psychiatry
Ghatak S, Dolatabadi N, Trudler D, et al. (2019) Mechanisms of hyperexcitability in Alzheimer's disease hiPSC-derived neurons and cerebral organoids vs. isogenic control. Elife. 8
Nakamura T, Lipton SA. (2019) Nitric Oxide-Dependent Protein Posttranslational Modifications Impair Mitochondrial Function and Metabolism to Contribute to Neurodegenerative Diseases. Antioxidants & Redox Signaling
Okamoto SI, Prikhodko O, Pina-Crespo J, et al. (2019) NitroSynapsin for the treatment of neurological manifestations of tuberous sclerosis complex in a rodent model. Neurobiology of Disease
Trudler D, Lipton SA. (2019) Novel Direct Conversion of Microglia to Neurons. Trends in Molecular Medicine
Ghatak S, Dolatabadi N, Trudler D, et al. (2019) Author response: Mechanisms of hyperexcitability in Alzheimer’s disease hiPSC-derived neurons and cerebral organoids vs isogenic controls Elife
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