Philip C. Wong, PhD

Affiliations: 
Pathology Johns Hopkins University, Baltimore, MD 
Area:
molecular mechanisms of neurodegenerative diseases
Website:
http://www.alzresearch.org/team.cfm?ID=29
Google:
"Philip Wong"
Bio:

http://pds17.pathology.jhmi.edu/N/n.web?EP=N&URL=/MCGI/SEND^WEBUTLTY(1058)/1543522402

Mean distance: 16.01 (cluster 32)
 
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Publications

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Chen L, Wei Z, Chan KW, et al. (2020) D-Glucose uptake and clearance in the tauopathy Alzheimer's disease mouse brain detected by on-resonance variable delay multiple pulse MRI. Journal of Cerebral Blood Flow and Metabolism : Official Journal of the International Society of Cerebral Blood Flow and Metabolism. 271678X20941264
Donde A, Sun M, Ling JP, et al. (2019) Splicing repression is a major function of TDP-43 in motor neurons. Acta Neuropathologica
Donde A, Sun M, Jeong YH, et al. (2019) Upregulation of ATG7 Attenuates Motor Neuron Dysfunction Associated with Depletion of TARDBP/TDP-43. Autophagy
Chen L, Wei Z, Chan K, et al. (2018) Protein aggregation linked to Alzheimer's disease revealed by saturation transfer MRI. Neuroimage
Donde A, Wong PC, Chen LL. (2017) Challenges and Advances in Gene Therapy Approaches for Neurodegenerative Disorders. Current Gene Therapy
Sun M, Bell W, LaClair KD, et al. (2017) Cryptic exon incorporation occurs in Alzheimer's brain lacking TDP-43 inclusion but exhibiting nuclear clearance of TDP-43. Acta Neuropathologica
Jeong YH, Ling JP, Lin SZ, et al. (2017) Tdp-43 cryptic exons are highly variable between cell types. Molecular Neurodegeneration. 12: 13
Bedont JL, LeGates TA, Buhr E, et al. (2016) An LHX1-Regulated Transcriptional Network Controls Sleep/Wake Coupling and Thermal Resistance of the Central Circadian Clockworks. Current Biology : Cb
Le NT, Chang L, Kovlyagina I, et al. (2016) Motor neuron disease, TDP-43 pathology, and memory deficits in mice expressing ALS-FTD-linked UBQLN2 mutations. Proceedings of the National Academy of Sciences of the United States of America
LaClair KD, Donde A, Ling JP, et al. (2016) Depletion of TDP-43 decreases fibril and plaque β-amyloid and exacerbates neurodegeneration in an Alzheimer's mouse model. Acta Neuropathologica
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