Brendan N. Lilley, Ph.D.
Affiliations: | Molecular and Cellular Biology | Harvard University, Cambridge, MA, United States |
Area:
Neural developmentGoogle:
"Brendan Lilley"Mean distance: 14.91 (cluster 11) | S | N | B | C | P |
Cross-listing: Chemistry Tree - Cell Biology Tree
Parents
Sign in to add mentorJoshua R. Sanes | post-doc | 2005- | Center for Brain Science, Harvard University |
Collaborators
Sign in to add collaboratorFranck Polleux | collaborator | 2006- | Center for Brain Science, Harvard University |
BETA: Related publications
See more...
Publications
You can help our author matching system! If you notice any publications incorrectly attributed to this author, please sign in and mark matches as correct or incorrect. |
Samuel MA, Voinescu PE, Lilley BN, et al. (2014) LKB1 and AMPK regulate synaptic remodeling in old age. Nature Neuroscience. 17: 1190-7 |
Lilley BN, Krishnaswamy A, Wang Z, et al. (2014) SAD kinases control the maturation of nerve terminals in the mammalian peripheral and central nervous systems. Proceedings of the National Academy of Sciences of the United States of America. 111: 1138-43 |
Lai LP, Lilley BN, Sanes JR, et al. (2013) Lkb1/Stk11 regulation of mTOR signaling controls the transition of chondrocyte fates and suppresses skeletal tumor formation. Proceedings of the National Academy of Sciences of the United States of America. 110: 19450-5 |
Nie J, Liu X, Lilley BN, et al. (2013) SAD-A kinase controls islet β-cell size and function as a mediator of mTORC1 signaling. Proceedings of the National Academy of Sciences of the United States of America. 110: 13857-62 |
Lilley BN, Pan YA, Sanes JR. (2013) SAD kinases sculpt axonal arbors of sensory neurons through long- and short-term responses to neurotrophin signals. Neuron. 79: 39-53 |
Nie J, Lilley BN, Pan YA, et al. (2013) SAD-A potentiates glucose-stimulated insulin secretion as a mediator of glucagon-like peptide 1 response in pancreatic β cells. Molecular and Cellular Biology. 33: 2527-34 |
Dougan SK, Hu CC, Paquet ME, et al. (2011) Derlin-2-deficient mice reveal an essential role for protein dislocation in chondrocytes. Molecular and Cellular Biology. 31: 1145-59 |
Kim JS, Lilley BN, Zhang C, et al. (2008) A chemical-genetic strategy reveals distinct temporal requirements for SAD-1 kinase in neuronal polarization and synapse formation. Neural Development. 3: 23 |
Barnes AP, Lilley BN, Pan YA, et al. (2007) LKB1 and SAD kinases define a pathway required for the polarization of cortical neurons. Cell. 129: 549-63 |
Mueller B, Lilley BN, Ploegh HL. (2006) SEL1L, the homologue of yeast Hrd3p, is involved in protein dislocation from the mammalian ER. The Journal of Cell Biology. 175: 261-70 |