Max Koppers, Ph.D.
Affiliations: | 2010-2016 | Translational Neuroscience | Utrecht University, Utrecht, Netherlands |
| 2016- | Physiology, Development and Neuroscience | University of Cambridge, Cambridge, England, United Kingdom |
Area:
Neurodegeneration, Axon guidance, Developmental Neuroscience, Local translationGoogle:
"Max Koppers"Mean distance: (not calculated yet)
Parents
Sign in to add mentor| Jeroen Pasterkamp | grad student | 2010-2016 | Cambridge |
| Christine E. Holt | post-doc | 2016- |
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Publications
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| Koppers M, Özkan N, Nguyen HH, et al. (2024) Axonal endoplasmic reticulum tubules control local translation via P180/RRBP1-mediated ribosome interactions. Developmental Cell |
| Koppers M, Holt CE. (2022) Receptor-Ribosome Coupling: A Link Between Extrinsic Signals and mRNA Translation in Neuronal Compartments. Annual Review of Neuroscience |
| Özkan N, Koppers M, van Soest I, et al. (2021) ER - lysosome contacts at a pre-axonal region regulate axonal lysosome availability. Nature Communications. 12: 4493 |
| Koppers M, Farías GG. (2021) Organelle distribution in neurons: Logistics behind polarized transport. Current Opinion in Cell Biology. 71: 46-54 |
| Shigeoka T, Koppers M, Wong HH, et al. (2019) On-Site Ribosome Remodeling by Locally Synthesized Ribosomal Proteins in Axons. Cell Reports. 29: 3605-3619.e10 |
| Koppers M, Cagnetta R, Shigeoka T, et al. (2019) Receptor-specific interactome as a hub for rapid cue-induced selective translation in axons. Elife. 8 |
| Koppers M, Cagnetta R, Shigeoka T, et al. (2019) Author response: Receptor-specific interactome as a hub for rapid cue-induced selective translation in axons Elife |
| Cioni JM, Lin JQ, Holtermann AV, et al. (2018) Late Endosomes Act as mRNA Translation Platforms and Sustain Mitochondria in Axons. Cell |
| Cioni JM, Koppers M, Holt CE. (2018) Molecular control of local translation in axon development and maintenance. Current Opinion in Neurobiology. 51: 86-94 |
| van Rheenen W, Shatunov A, Dekker AM, et al. (2016) Genome-wide association analyses identify new risk variants and the genetic architecture of amyotrophic lateral sclerosis. Nature Genetics. 48: 1043-8 |