Edward Giniger

NINDS National Institutes of Health, Bethesda, MD 
"Edward Giniger"
Mean distance: 13.29 (cluster 11)


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Mark Ptashne grad student Harvard (Chemistry Tree)
Lily Y. Jan post-doc UCSF
Yuh Nung Jan post-doc UCSF


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Kristina A. McLinden post-doc 2012- NIH
Ramakrishnan Kannan post-doc 2007-2015
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Spurrier J, Shukla AK, Buckley T, et al. (2019) Expression of a Fragment of Ankyrin 2 Disrupts the Structure of the Axon Initial Segment and Causes Axonal Degeneration in Drosophila. Molecular Neurobiology
Shukla AK, Spurrier J, Kuzina I, et al. (2019) Hyperactive Innate Immunity Causes Degeneration of Dopamine Neurons upon Altering Activity of Cdk5. Cell Reports. 26: 131-144.e4
Kannan R, Cox E, Wang L, et al. (2018) Tyrosine phosphorylation and proteolytic cleavage of Notch are required for non-canonical Notch/Abl signaling in Drosophila axon guidance. Development (Cambridge, England). 145
Kannan R, Giniger E. (2017) New perspectives on the roles of Abl tyrosine kinase in axon patterning. Fly. 0
Kannan R, Song JK, Karpova T, et al. (2017) The Abl pathway bifurcates to balance Enabled and Rac signaling in axon patterning in Drosophila. Development (Cambridge, England)
Smith-Trunova S, Prithviraj R, Spurrier J, et al. (2015) Cdk5 regulates developmental remodeling of mushroom body neurons in Drosophila. Developmental Dynamics : An Official Publication of the American Association of Anatomists
Kannan R, Kuzina I, Wincovitch S, et al. (2014) The Abl/enabled signaling pathway regulates Golgi architecture in Drosophila photoreceptor neurons. Molecular Biology of the Cell. 25: 2993-3005
Ferreira T, Ou Y, Li S, et al. (2014) Dendrite architecture organized by transcriptional control of the F-actin nucleator Spire. Development (Cambridge, England). 141: 650-60
Kotlyanskaya L, McLinden KA, Giniger E. (2013) Of proneurotrophins and their antineurotrophic effects. Science Signaling. 6: pe6
McLinden KA, Trunova S, Giniger E. (2012) At the Fulcrum in Health and Disease: Cdk5 and the Balancing Acts of Neuronal Structure and Physiology. Brain Disorders & Therapy. 2012: 001
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