Tadafumi Hashimoto

2012-2021 The University of Tokyo (Japan) 
 2021- National Center of Neurology and Psychiatry, Tokyo, Kodaira-shi, Tōkyō-to, Japan 
Alzheimer's disease, Neurodegenerative disorders
"Tadafumi Hashimoto"
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Watanabe H, Murakami R, Tsumagari K, et al. (2023) Astrocytic APOE4 genotype-mediated negative impacts on synaptic architecture in human pluripotent stem cell model. Stem Cell Reports. 18: 1854-1869
Kishino Y, Matsukawa K, Matsumoto T, et al. (2022) Casein kinase 1δ/ε phosphorylates fused in sarcoma (FUS) and ameliorates FUS-mediated neurodegeneration. The Journal of Biological Chemistry. 102191
Kaneshiro N, Komai M, Imaoka R, et al. (2022) Lipid flippase dysfunction as a therapeutic target for endosomal anomalies in Alzheimer's disease. Iscience. 25: 103869
Ishida K, Yamada K, Nishiyama R, et al. (2022) Glymphatic system clears extracellular tau and protects from tau aggregation and neurodegeneration. The Journal of Experimental Medicine. 219
An H, Litscher G, Watanabe N, et al. (2021) ALS-linked cytoplasmic FUS assemblies are compositionally different from physiological stress granules and sequester hnRNPA3, a novel modifier of FUS toxicity. Neurobiology of Disease. 162: 105585
Matsukawa K, Kukharsky MS, Park SK, et al. (2021) Long non-coding RNA NEAT1_1 ameliorates TDP-43 toxicity in in vivo models of TDP-43 proteinopathy. Rna Biology. 1-9
Hashimoto T, Fujii D, Naka Y, et al. (2020) Collagenous Alzheimer amyloid plaque component impacts on the compaction of amyloid-β plaques. Acta Neuropathologica Communications. 8: 212
Abe Y, Ikegawa N, Yoshida K, et al. (2020) Behavioral and electrophysiological evidence for a neuroprotective role of aquaporin-4 in the 5xFAD transgenic mice model. Acta Neuropathologica Communications. 8: 67
Munezane H, Oizumi H, Wakabayashi T, et al. (2019) Roles of Collagen XXV and Its Putative Receptors PTPσ/δ in Intramuscular Motor Innervation and Congenital Cranial Dysinnervation Disorder. Cell Reports. 29: 4362-4376.e6
Park S, Park SK, Watanabe N, et al. (2019) Calcium-responsive transactivator (CREST) toxicity is rescued by loss of PBP1/ATXN2 function in a novel yeast proteinopathy model and in transgenic flies. Plos Genetics. 15: e1008308
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