Biljana Djukic, Ph.D. - Publications

Affiliations: 
University of North Carolina, Chapel Hill, Chapel Hill, NC 
Area:
astrocyte

8 high-probability publications. We are testing a new system for linking publications to authors. You can help! If you notice any inaccuracies, please sign in and mark papers as correct or incorrect matches. If you identify any major omissions or other inaccuracies in the publication list, please let us know.

Year Citation  Score
2024 Maeda S, Djukic B, Taneja P, Yu GQ, Lo I, Davis A, Craft R, Guo W, Wang X, Kim D, Ponnusamy R, Gill TM, Masliah E, Mucke L. Author Correction: Expression of A152T human tau causes age-dependent neuronal dysfunction and loss in transgenic mice. Embo Reports. PMID 39363060 DOI: 10.1038/s44319-024-00212-8  0.343
2017 Krabbe G, Minami SS, Etchegaray JI, Taneja P, Djukic B, Davalos D, Le D, Lo I, Zhan L, Reichert MC, Sayed F, Merlini M, Ward ME, Perry DC, Lee SE, et al. Microglial NFκB-TNFα hyperactivation induces obsessive-compulsive behavior in mouse models of progranulin-deficient frontotemporal dementia. Proceedings of the National Academy of Sciences of the United States of America. PMID 28438992 DOI: 10.1073/Pnas.1700477114  0.35
2016 Maeda S, Djukic B, Taneja P, Yu GQ, Lo I, Davis A, Craft R, Guo W, Wang X, Kim D, Ponnusamy R, Gill TM, Masliah E, Mucke L. Expression of A152T human tau causes age-dependent neuronal dysfunction and loss in transgenic mice. Embo Reports. PMID 26931567 DOI: 10.15252/Embr.201541438  0.368
2015 Suberbielle E, Djukic B, Evans M, Kim DH, Taneja P, Wang X, Finucane M, Knox J, Ho K, Devidze N, Masliah E, Mucke L. DNA repair factor BRCA1 depletion occurs in Alzheimer brains and impairs cognitive function in mice. Nature Communications. 6: 8897. PMID 26615780 DOI: 10.1038/Ncomms9897  0.305
2014 Tong LM, Djukic B, Arnold C, Gillespie AK, Yoon SY, Wang MM, Zhang O, Knoferle J, Rubenstein JL, Alvarez-Buylla A, Huang Y. Inhibitory interneuron progenitor transplantation restores normal learning and memory in ApoE4 knock-in mice without or with Aβ accumulation. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 34: 9506-15. PMID 25031394 DOI: 10.1523/Jneurosci.0693-14.2014  0.331
2010 Chever O, Djukic B, McCarthy KD, Amzica F. Implication of Kir4.1 channel in excess potassium clearance: an in vivo study on anesthetized glial-conditional Kir4.1 knock-out mice. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 30: 15769-77. PMID 21106816 DOI: 10.1523/Jneurosci.2078-10.2010  0.513
2007 Djukic B, Casper KB, Philpot BD, Chin LS, McCarthy KD. Conditional knock-out of Kir4.1 leads to glial membrane depolarization, inhibition of potassium and glutamate uptake, and enhanced short-term synaptic potentiation. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 27: 11354-65. PMID 17942730 DOI: 10.1523/Jneurosci.0723-07.2007  0.507
2007 Wiencken-Barger AE, Djukic B, Casper KB, McCarthy KD. A role for Connexin43 during neurodevelopment. Glia. 55: 675-86. PMID 17311295 DOI: 10.1002/Glia.20484  0.478
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