| Year |
Citation |
Score |
| 2017 |
Flinspach M, Xu Q, Piekarz AD, Fellows R, Hagan R, Gibbs A, Liu Y, Neff RA, Freedman J, Eckert WA, Zhou M, Bonesteel R, Pennington MW, Eddinger KA, Yaksh TL, et al. Insensitivity to pain induced by a potent selective closed-state Nav1.7 inhibitor. Scientific Reports. 7: 39662. PMID 28045073 DOI: 10.1038/srep39662 |
0.377 |
|
| 2014 |
Tan ZY, Piekarz AD, Priest BT, Knopp KL, Krajewski JL, McDermott JS, Nisenbaum ES, Cummins TR. Tetrodotoxin-resistant sodium channels in sensory neurons generate slow resurgent currents that are enhanced by inflammatory mediators. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 34: 7190-7. PMID 24849353 DOI: 10.1523/Jneurosci.5011-13.2014 |
0.702 |
|
| 2012 |
King AM, Yang XF, Wang Y, Dustrude ET, Barbosa C, Due MR, Piekarz AD, Wilson SM, White FA, Salomé C, Cummins TR, Khanna R, Kohn H. Identification of the benzyloxyphenyl pharmacophore: a structural unit that promotes sodium channel slow inactivation. Acs Chemical Neuroscience. 3: 1037-49. PMID 23259039 DOI: 10.1021/Cn300129D |
0.611 |
|
| 2012 |
Due MR, Piekarz AD, Wilson N, Feldman P, Ripsch MS, Chavez S, Yin H, Khanna R, White FA. Neuroexcitatory effects of morphine-3-glucuronide are dependent on Toll-like receptor 4 signaling. Journal of Neuroinflammation. 9: 200. PMID 22898544 DOI: 10.1186/1742-2094-9-200 |
0.453 |
|
| 2012 |
Piekarz AD, Due MR, Khanna M, Wang B, Ripsch MS, Wang R, Meroueh SO, Vasko MR, White FA, Khanna R. CRMP-2 peptide mediated decrease of high and low voltage-activated calcium channels, attenuation of nociceptor excitability, and anti-nociception in a model of AIDS therapy-induced painful peripheral neuropathy. Molecular Pain. 8: 54. PMID 22828369 DOI: 10.1186/1744-8069-8-54 |
0.394 |
|
| 2012 |
Song W, Xiao Y, Chen H, Ashpole NM, Piekarz AD, Ma P, Hudmon A, Cummins TR, Shou W. The human Nav1.5 F1486 deletion associated with long QT syndrome leads to impaired sodium channel inactivation and reduced lidocaine sensitivity. The Journal of Physiology. 590: 5123-39. PMID 22826127 DOI: 10.1113/Jphysiol.2012.235374 |
0.697 |
|
| 2011 |
Wilson SM, Brittain JM, Piekarz AD, Ballard CJ, Ripsch MS, Cummins TR, Hurley JH, Khanna M, Hammes NM, Samuels BC, White FA, Khanna R. Further insights into the antinociceptive potential of a peptide disrupting the N-type calcium channel-CRMP-2 signaling complex. Channels (Austin, Tex.). 5: 449-56. PMID 21829088 DOI: 10.4161/Chan.5.5.17363 |
0.692 |
|
| 2011 |
Theile JW, Jarecki BW, Piekarz AD, Cummins TR. Nav1.7 mutations associated with paroxysmal extreme pain disorder, but not erythromelalgia, enhance Navbeta4 peptide-mediated resurgent sodium currents. The Journal of Physiology. 589: 597-608. PMID 21115638 DOI: 10.1113/Jphysiol.2010.200915 |
0.665 |
|
| 2010 |
Jarecki BW, Piekarz AD, Jackson JO, Cummins TR. Human voltage-gated sodium channel mutations that cause inherited neuronal and muscle channelopathies increase resurgent sodium currents. The Journal of Clinical Investigation. 120: 369-78. PMID 20038812 DOI: 10.1172/Jci40801 |
0.708 |
|
| 2009 |
Brittain JM, Piekarz AD, Wang Y, Kondo T, Cummins TR, Khanna R. An atypical role for collapsin response mediator protein 2 (CRMP-2) in neurotransmitter release via interaction with presynaptic voltage-gated calcium channels. The Journal of Biological Chemistry. 284: 31375-90. PMID 19755421 DOI: 10.1074/Jbc.M109.009951 |
0.609 |
|
| 2009 |
Jarecki BW, Piekarz AD, Jackson JO, Cummins TR. Impact Of NaV1.7-PEPD Missense Mutations That Slow The Rate Of Inactivation On Sensory Neuronal Resurgent Sodium Currents Biophysical Journal. 96: 663a. DOI: 10.1016/J.Bpj.2008.12.3506 |
0.691 |
|
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