| Year |
Citation |
Score |
| 2022 |
Ayers JI, Lee J, Monteiro O, Woerman AL, Lazar AA, Condello C, Paras NA, Prusiner SB. Different α-synuclein prion strains cause dementia with Lewy bodies and multiple system atrophy. Proceedings of the National Academy of Sciences of the United States of America. 119. PMID 35115402 DOI: 10.1073/pnas.2113489119 |
0.354 |
|
| 2020 |
Ayers JI, Borchelt DR. Phenotypic diversity in ALS and the role of poly-conformational protein misfolding. Acta Neuropathologica. PMID 32930869 DOI: 10.1007/S00401-020-02222-X |
0.31 |
|
| 2020 |
Ayers JI, Paras NA, Prusiner SB. Expanding spectrum of prion diseases. Emerging Topics in Life Sciences. 4: 155-167. PMID 32803268 DOI: 10.1042/ETLS20200037 |
0.315 |
|
| 2020 |
Ayers JI, Prusiner SB. Prion protein - mediator of toxicity in multiple proteinopathies. Nature Reviews. Neurology. PMID 32123368 DOI: 10.1038/s41582-020-0332-8 |
0.317 |
|
| 2020 |
Crown A, McAlary L, Fagerli E, Brown H, Yerbury JJ, Galaleldeen A, Cashman NR, Borchelt DR, Ayers JI. Tryptophan residue 32 in human Cu-Zn superoxide dismutase modulates prion-like propagation and strain selection. Plos One. 15: e0227655. PMID 31999698 DOI: 10.1371/Journal.Pone.0227655 |
0.388 |
|
| 2019 |
Crown AM, Roberts BL, Crosby K, Brown H, Ayers JI, Hart PJ, Borchelt DR. Experimental Mutations in Superoxide Dismutase 1 Provide Insight into Potential Mechanisms Involved in Aberrant Aggregation in Familial Amyotrophic Lateral Sclerosis. G3 (Bethesda, Md.). PMID 30622123 DOI: 10.1534/G3.118.200787 |
0.333 |
|
| 2018 |
Crosby K, Crown AM, Roberts BL, Brown H, Ayers JI, Borchelt DR. Loss of charge mutations in solvent exposed Lys residues of superoxide dismutase 1 do not induce inclusion formation in cultured cell models. Plos One. 13: e0206751. PMID 30399166 DOI: 10.1371/Journal.Pone.0206751 |
0.31 |
|
| 2018 |
Ayers JI, Riffe CJ, Sorrentino ZA, Diamond J, Fagerli E, Brooks M, Galaleldeen A, Hart PJ, Giasson BI. Localized induction of wild-type and mutant alpha-synuclein aggregation reveals propagation along neuroanatomical tracts. Journal of Virology. PMID 29976670 DOI: 10.1128/Jvi.00586-18 |
0.338 |
|
| 2016 |
Ayers JI, McMahon B, Gill S, Lelie HL, Fromholt S, Brown H, Valentine JS, Whitelegge JP, Borchelt DR. Relationship between mutant SOD1 maturation and inclusion formation in cell models. Journal of Neurochemistry. PMID 27727458 DOI: 10.1111/Jnc.13864 |
0.363 |
|
| 2016 |
Ayers JI, Diamond J, Sari A, Fromholt S, Galaleldeen A, Ostrow LW, Glass JD, Hart PJ, Borchelt DR. Distinct conformers of transmissible misfolded SOD1 distinguish human SOD1-FALS from other forms of familial and sporadic ALS. Acta Neuropathologica. PMID 27704280 DOI: 10.1007/S00401-016-1623-4 |
0.322 |
|
| 2015 |
Ayers JI, Fromholt SE, O'Neal VM, Diamond JH, Borchelt DR. Prion-like propagation of mutant SOD1 misfolding and motor neuron disease spread along neuroanatomical pathways. Acta Neuropathologica. PMID 26650262 DOI: 10.1007/S00401-015-1514-0 |
0.317 |
|
| 2015 |
Xu G, Fromholt S, Ayers JI, Brown H, Siemienski Z, Crosby KW, Mayer CA, Janus C, Borchelt DR. Substantially elevating the levels of αB-crystallin in spinal motor neurons of mutant SOD1 mice does not significantly delay paralysis or attenuate mutant protein aggregation. Journal of Neurochemistry. 133: 452-64. PMID 25557022 DOI: 10.1111/Jnc.13022 |
0.35 |
|
| 2015 |
Xu G, Ayers JI, Roberts BL, Brown H, Fromholt S, Green C, Borchelt DR. Direct and indirect mechanisms for wild-type SOD1 to enhance the toxicity of mutant SOD1 in bigenic transgenic mice. Human Molecular Genetics. 24: 1019-35. PMID 25305079 DOI: 10.1093/Hmg/Ddu517 |
0.305 |
|
| 2014 |
Ayers JI, Fromholt S, Koch M, DeBosier A, McMahon B, Xu G, Borchelt DR. Experimental transmissibility of mutant SOD1 motor neuron disease. Acta Neuropathologica. 128: 791-803. PMID 25262000 DOI: 10.1007/S00401-014-1342-7 |
0.31 |
|
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